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Titlebook: Aging Mechanisms II; Longevity, Metabolis Nozomu Mori Book 2022 The Editor(s) (if applicable) and The Author(s), under exclusive license to

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發(fā)表于 2025-3-21 19:18:59 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Aging Mechanisms II
期刊簡稱Longevity, Metabolis
影響因子2023Nozomu Mori
視頻videohttp://file.papertrans.cn/152/151455/151455.mp4
發(fā)行地址Summarizes the latest research on aging biology from leading laboratories.Discusses biological mechanisms of aging from molecular and cellular levels to physiological and systemic levels.Covers import
圖書封面Titlebook: Aging Mechanisms II; Longevity, Metabolis Nozomu Mori Book 2022 The Editor(s) (if applicable) and The Author(s), under exclusive license to
影響因子.This book describes the recent advancement of basic research on the biology of aging and longevity studies in various organisms, as well as the neurobiology of aging and neurodegeneration mechanisms. Chapters present new findings and conceptual developments concerning the basic mechanisms of aging and longevity determination. .As. a follow-up volume to the previous book .Aging Mechanisms. (2015), it overviews the rapid progress of aging research introducing new topics from leading laboratories in Japan.? ..Chapter contributors are selected based on recent scientific achievements on the mechanisms of aging in various model organisms, including yeast, worm (.C. elegans.), fly (.Drosophila.), mice, and rats. Chapters are ordered from the discussion on molecular and cellular levels to physiological and systemic levels. The book also provides an overview of aging science in the region and helps readers quickly grasp who is doing what in this research area...As the aging of population becomes an ever more pressing issue in Asia, advancing the understanding of basic mechanisms of organism aging and longevity determination will be crucial to developing more effective therapies and protect
Pindex Book 2022
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,Das Leben vor der Dichterzeit (1825–1865), found that N-terminal pro-B-type natriuretic peptide (NT-proBNP), interleukin-6, cystatin C, cholinesterase, and albumin were associated with all-cause mortality. Of these, low NT-proBNP levels were strongly associated with survival beyond 105?years, while albumin levels were associated with high m
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Die Conrad Ferdinand Meyer-Forschung,nscription of various genes and consequently the life span of . will change. In this chapter, we will present the basic characteristics of ., classical and up-to-date methods of measuring life span, aging phenotype using fluorescence and well-known longevity genes. This article will convince you tha
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An Unsolved Problem in Gerontology Yet: Molecular Mechanisms of Biological Aging—A Historical and Crthe free radical or oxidative theory, was proposed more than half a century ago but has recently faced severe criticisms to which I shall refer. So far, no single theory has been able to successfully explain the mechanism of biological aging. We are thus awaiting emergence of a new paradigm or an in
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Clinical and Basic Biology of Werner Syndrome, the Model Disease of Human Aginghortened telomeres, chronic inflammation due to cellular senescence- and senescence-associated secretory phenotype (SASP), decreased mitochondrial function and accumulation of oxidative stress, stem cell senescence, and epigenetic changes. While most premature aging syndromes occur in childhood and
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