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Titlebook: Viral Gastroenteritis; Shunzo Chiba,Mary K. Estes,Charles H. Calisher Conference proceedings 1996 Springer-Verlag Wien 1996 antibody.antig

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發(fā)表于 2025-3-21 19:46:30 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Viral Gastroenteritis
編輯Shunzo Chiba,Mary K. Estes,Charles H. Calisher
視頻videohttp://file.papertrans.cn/984/983189/983189.mp4
叢書名稱Archives of Virology. Supplementa
圖書封面Titlebook: Viral Gastroenteritis;  Shunzo Chiba,Mary K. Estes,Charles H. Calisher Conference proceedings 1996 Springer-Verlag Wien 1996 antibody.antig
描述Recently, rapid developments have occurred in the field of viral gastroenteritis. This book is an update of fundamental and practical aspects of viral gastroenteritis. Among the various agents that cause viral gastroenteritis, group A rotaviruses and caliciviruses are the focus of this volume because of their clinical impact and the significance of new findings about them.
出版日期Conference proceedings 1996
關鍵詞antibody; antigen; infection; molecular biology; pathogenesis; vaccination; vaccine; virus
版次1
doihttps://doi.org/10.1007/978-3-7091-6553-9
isbn_softcover978-3-211-82875-5
isbn_ebook978-3-7091-6553-9Series ISSN 0939-1983
issn_series 0939-1983
copyrightSpringer-Verlag Wien 1996
The information of publication is updating

書目名稱Viral Gastroenteritis影響因子(影響力)




書目名稱Viral Gastroenteritis影響因子(影響力)學科排名




書目名稱Viral Gastroenteritis網絡公開度




書目名稱Viral Gastroenteritis網絡公開度學科排名




書目名稱Viral Gastroenteritis被引頻次




書目名稱Viral Gastroenteritis被引頻次學科排名




書目名稱Viral Gastroenteritis年度引用




書目名稱Viral Gastroenteritis年度引用學科排名




書目名稱Viral Gastroenteritis讀者反饋




書目名稱Viral Gastroenteritis讀者反饋學科排名




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,Identification of the minimal replicase and the minimal promoter of (—)-strand synthesis, functionae activity, to the 5′-terminal 27 nucleotides (nt 1–27) and the 3′-terminal 26 nucleotides (nt 1037-1062). Further analysis showed that a minimal promoter of (—)-strand synthesis was contained in the 3′-terminal 7 nucleotides (nt 1056-1062); the sequence conserved at the 3′-terminus of all rotavirus
地板
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Rotavirus protein expression is important for virus assembly and pathogenesis,termine the mechanism by which NSP4 causes an increase in [Ca.]i showed that Ca. is released from a subset of the thapsigargin-sensitive store [endoplasmic reticulum (ER)]. However, exogenously added and endogenously expressed NSP4 use different mechanisms to alter the Ca. permeability of the ER mem
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Development of rotavirus molecular epidemiology: electropherotyping,uman rotavirus strain variation. Since then, technical improvements have greatly increased the sensitivity of the procedures, and electropherotyping has been recognized as a powerful and economical method for epidemiological studies of rotaviruses.
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VP4 and VP7 typing using monoclonal antibodies,ular VP7 serotype, it is necessary to type with a panel of N-MAbs specific for that serotype..N-MAbs to VP4 of human rotavirus are difficult to raise and few have proven suitable for VP4 serotyping by EIA. The specificity of the assay for each P type is highest when the VP7 serotype specificity of t
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The gnotobiotic piglet as a model for studies of disease pathogenesis and immunity to human rotavirtic piglets in our laboratory have confirmed that villous atrophy is induced in piglets given virulent but not cell culture attenuated human rotavirus (G1, P1 A, Wa strain) and have revealed that factors other than villous atrophy may contribute to the early diarrhea induced. A comprehensive examina
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