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Titlebook: Viral Entry into Host Cells; Stefan P?hlmann,Graham Simmons Book 2013 The Editor(s) (if applicable) and The Author(s), under exclusive lic

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發(fā)表于 2025-3-21 18:57:06 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Viral Entry into Host Cells
編輯Stefan P?hlmann,Graham Simmons
視頻videohttp://file.papertrans.cn/984/983186/983186.mp4
概述Provides a comprehensive overview of interplay between viral and cellular factors during virus entry.Reviews key processes underlying host cell entry of viruses.Discusses cellular entry of reoviruses
叢書(shū)名稱(chēng)Advances in Experimental Medicine and Biology
圖書(shū)封面Titlebook: Viral Entry into Host Cells;  Stefan P?hlmann,Graham Simmons Book 2013 The Editor(s) (if applicable) and The Author(s), under exclusive lic
描述For a virus to invade a host cell it needs to penetrate the physical barrier imposed by the plasma membrane. Viruses have evolved specialized surface proteins to meet this challenge. These proteins facilitate delivery of the viral genetic information into the host cell by either fusing the viral envelope with a host cell membrane or by forming membrane pores. Membrane fusion and pore formation critically depend on the engagement of host cell receptors and receptor choice is a key determinant of viral tropism. The multi-faceted interplay between viral and cellular factors during virus entry is a fascinating field of study, which can provide important insight into viral pathogenesis and define new targets for intervention. This book provides a comprehensive overview of this exciting field of research.
出版日期Book 2013
關(guān)鍵詞Apoptosis; Host cell; Reoviruses; Viral entry
版次1
doihttps://doi.org/10.1007/978-1-4614-7651-1
isbn_ebook978-1-4614-7651-1Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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Reovirus Receptors, Cell Entry, and Proapoptotic Signaling,type of carbohydrate bound varying by serotype. Following attachment to JAM-A and carbohydrate, reovirus internalization is mediated by β1 integrins, most likely via clathrin-dependent endocytosis. In the endocytic compartment, reovirus outer-capsid protein σ3 is removed by acid-dependent cysteine p
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Cellular Entry of Retroviruses,iruses, enter target cells, and how the interaction of the viral and cellular factors involved in the cellular entry of these viruses impacts viral tropism, pathogenesis and approaches to therapy and vaccine development.
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Class II Fusion Proteins,ansmembrane anchor) against each other, resulting in membrane fusion. In class II fusion proteins, the fold-back is triggered by the reduced pH of an endosome, and is accompanied by the assembly of fusion protein monomers into trimers. The fold-back occurs by domain rearrangement rather than by an e
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