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Titlebook: Urea Cycle Diseases; A. Lowenthal,A. Mori,B. Marescau Book 1982 The Editor(s) (if applicable) and The Author(s), under exclusive license t

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發(fā)表于 2025-3-21 18:32:40 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Urea Cycle Diseases
編輯A. Lowenthal,A. Mori,B. Marescau
視頻videohttp://file.papertrans.cn/945/944193/944193.mp4
叢書名稱Advances in Experimental Medicine and Biology
圖書封面Titlebook: Urea Cycle Diseases;  A. Lowenthal,A. Mori,B. Marescau Book 1982 The Editor(s) (if applicable) and The Author(s), under exclusive license t
描述Introduction New Facets in Urea Cycle Disorders INTRODUCTION A. Lowenthal Laboratory of Neurochemistry, Born-Bunge Foundation, Universitaire Instelling Antwerpen, Wilrijk, Belgium This occasion is by no means the first meeting devoted to urea cycle diseases. It has been preceeded by meetings held in the Netherlands and in Spain. Accordingly the justification for a further meeting is not immediately evident. The reason for it is that the problems related to urea cycle diseases are developing fast, as instanced inter alia by I) the relation observed between hyperornithinemia and gyrate atrophy and the therapeutic acquisitions which result from it 2) the treatment of hyperammonemias. If the diagnosis of urea cycle disease is easily established by following standard principles and techniques, i.e. by means of amino acid analysis, with or without prior loading tests, by ammonemia measurement and by enzyme determination, also if these operations produce clear and precise conclusions in the matter of genetics and preventive medicine, yet many physiopathological questions remain unanswered and a number of therapeutic problems of these remain unsolved.
出版日期Book 1982
關(guān)鍵詞Glutamat; amino acid; chemistry; diagnosis; enzyme; enzymes; genetics; kidney; medicine; metabolism; mitochond
版次1
doihttps://doi.org/10.1007/978-1-4757-6903-6
isbn_softcover978-1-4757-6905-0
isbn_ebook978-1-4757-6903-6Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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發(fā)表于 2025-3-21 20:58:47 | 只看該作者
Book 1982and by enzyme determination, also if these operations produce clear and precise conclusions in the matter of genetics and preventive medicine, yet many physiopathological questions remain unanswered and a number of therapeutic problems of these remain unsolved.
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A. Lowenthalrls addressed), these differently named classifications are part of a prevalent, late-twentieth-century postfeminist discourse that blends a more mainstream — or commoditized (depending on your view/critique of popular culture) — and individualized version of feminism with a range of feminine and/or sexual markers.
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發(fā)表于 2025-3-22 09:04:51 | 只看該作者
A. Moricourses. is variously inscribed on and embodied by the figure of the female athlete. It therefore draws on an understanding of postfeminism as a ‘sensibility’. where notions of female empowerment are increasingly asserted through an emphasis on individualism, choice, consumption and (bodily) self-discipline.
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0065-2598 Instelling Antwerpen, Wilrijk, Belgium This occasion is by no means the first meeting devoted to urea cycle diseases. It has been preceeded by meetings held in the Netherlands and in Spain. Accordingly the justification for a further meeting is not immediately evident. The reason for it is that the
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New Facets in Urea Cycle Disordersby the discoverers themselves. Sparse-fur mutant mice have become a model for human ornithine transcarbamylase deficiency, and will contribute extensively to investigations of human diseases in the future.
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