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Titlebook: Surgical Pathology of Non-neoplastic Gastrointestinal Diseases; Lizhi Zhang,Vishal S. Chandan,Tsung-Teh Wu Book 2019 Springer Nature Switz

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樓主: inroad
21#
發(fā)表于 2025-3-25 04:26:00 | 只看該作者
Infectious Disorders of the Duodenum and Small Bowelwel infections of the duodenum and small intestine (jejunum and ileum). Infectious agents which cause disease that may result in a biopsy to obtain a diagnosis are covered in this chapter. Clinical features, histopathologic features, differential diagnosis, and treatment and prognosis are covered fo
22#
發(fā)表于 2025-3-25 11:20:21 | 只看該作者
Drug-Induced Injury, Polyps, Congenital, and Miscellaneous Disorders, Yttrium-90, and lanthanum can cause relatively specific histological findings in the duodenum. Benign duodenal polyps are composed of a heterogeneous group of lesions such as hamartomatous polyps, hyperplastic and inflammatory polyps, gastric and pancreatic heterotopia, Brunner gland hyperplasia,
23#
發(fā)表于 2025-3-25 13:29:33 | 只看該作者
24#
發(fā)表于 2025-3-25 19:54:29 | 只看該作者
Drug-Induced Injury, Vascular, Congenital, and Miscellaneous Disorders ileum is a bit different from that in duodenum and colon. Because of limited endoscopic accessibility and availability of tissue sample, many entities are less understood or recognized by pathologists. Besides Crohn’s disease, there are many nonneoplastic or congenital disorders in jejunum and ileu
25#
發(fā)表于 2025-3-25 23:53:49 | 只看該作者
Normal Histology of Gastrointestinal Tractntric layers progressing outward from the lumen, namely mucosa, submucosa, muscularis propria, and serosa or adventitia. Although the mucosal features vary significantly from one region to another, the other layers share many of the same features throughout the GI tract, albeit some differences do exist.
26#
發(fā)表于 2025-3-26 01:49:26 | 只看該作者
27#
發(fā)表于 2025-3-26 06:30:38 | 只看該作者
28#
發(fā)表于 2025-3-26 10:06:05 | 只看該作者
29#
發(fā)表于 2025-3-26 13:29:16 | 只看該作者
30#
發(fā)表于 2025-3-26 18:24:55 | 只看該作者
Eosinophilic Esophagitis: What is the definition of “increased eosinophils?” Should there be a marked increase before EGID is considered? What are the other conditions to be excluded, and are there histologic clues to those conditions? Consensus criteria are available for eosinophilic esophagitis and are reviewed here.
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