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Titlebook: Stunning, Hibernation, and Calcium in Myocardial Ischemia and Reperfusion; Lionel H. Opie (Professor of Medicine, Visiting Pr Book 1992 Kl

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書(shū)目名稱(chēng)Stunning, Hibernation, and Calcium in Myocardial Ischemia and Reperfusion
編輯Lionel H. Opie (Professor of Medicine, Visiting Pr
視頻videohttp://file.papertrans.cn/882/881127/881127.mp4
圖書(shū)封面Titlebook: Stunning, Hibernation, and Calcium in Myocardial Ischemia and Reperfusion;  Lionel H. Opie (Professor of Medicine, Visiting Pr Book 1992 Kl
描述Stunning is potentially an important complication of myocardialreperfusion. In contrast, hibernation is an important complication ofmyocardial ischemia. This book presents different viewpoints on thesesubjects, taken from a series of peer-reviewed articles which firstappeared in .Cardiovascular Drugs and Therapy., and now in thisbook. In addition, a certain number of articles were directly invitedfor the book. .The introductory article is by Eugene Braunwald, who with Klonerintroduced the term .myocardial stunning. in 1982. Theexperimental phenomenon had first been described by Heyndrickx .etal.. This important concept of stunning is examined from a number ofpoints of view in a series of articles which clarify the experimentalcauses and the clinical implications. The section on hibernation isintroduced by Tubau and Rahimtoola, the latter having first propoundedthe concept in 1989. .The difference between stunning and hibernation is that stunning isessentially a post-ischemic dysfunction of the myocardium, temporaryin nature, occurring at a time when coronary blood flow is apparentlynormal or supranormal. Hibernation, on the other hand, is a conditionof reduced contractile activity,
出版日期Book 1992
關(guān)鍵詞adenosine; blood; calcium; cardiovascular; complication; diagnosis; drugs; hand; heart; metabolism; therapy; to
版次1
doihttps://doi.org/10.1007/978-1-4613-1517-9
isbn_softcover978-0-7923-1793-7
isbn_ebook978-1-4613-1517-9
copyrightKluwer Academic Publishers 1992
The information of publication is updating

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Postischemic Stunning—The Case for Calcium as the Ultimate Culprited with cytosolic calcium overload and an apparently normal or nearly normal mechanical function. Agents enhancing calcium influx, if introduced at this stage, may worsen subsequent stunning, whereas those inhibiting calcium influx may lessen the extent of subsequent stunning. The second phase, true
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