Titlebook: Strategic Medical Science Against Brain Attack; Haruhiko Kikuchi (President Emeritus) Book 2002 Springer-Verlag Tokyo 2002 Brain protectio

兩種語(yǔ)言

Cerebrovascular disease and hypothermia Part I: effects of mild hypothermia in the management of cerin complications persist when hypothermic therapy is used in clinical settings such as head injury and stroke. While several clinical trials of hypothermic therapy have been performed, the efficacy of the hypothermia has not been definitively proven. The current paper describes the development and u

賞錢(qián)

Confidential

Neuroprotection by mild hypothermia for temporary or permanent focal ischemiarogression understanding the efficacy and specific characteristics of prolonged mild hypothermia for ischemic stroke in rats. Using temporary or permanent focal ischemia models, the rats were treated by mild (33 °C) hypothermia at different schedules. The targeted mild hypothermia was applied only i

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注視

Neural stem cells as a tool for cell transplantationd into neurons in the hippocampus. Furthermore, the rats with more than 120 neurons showed recovery from the impairment of spatial recognition in the water maze test..These results support the concept that NSCs in the adult brain have a potential to differentiate into functional neurons that can be

Chandelier

鄙視讀作

The MEK/ERK pathway as a target for stroke therapyay is crucial in neuronal damage resulting from ischemia and reperfusion or other related oxidative stresses. It is suggested that the MEK/ERK pathway may be a novel target for stroke treatment. This article will review the evidence that implicates the MEK/ERK pathway in cerebral ischemia and reperf

宿醉

Structure and functions of pituitary adenylate cyclase activating polypeptide (PACAP) as a neurotropPACAP appears to function as a neuroprotective factor that attenuates the neuronal damages resulting from various insults like focal ischemia by middle cerebral artery occlusion (MCAO). Together with the fact that PACAP can easily cross the blood brain barrier, these suggest that PACAP may be a prom

CLEAR

Brain ischemia and spreading depression in a primate model gradually thereafter. CMRglc at 24 h after embolization in these regions was increased compared with that in the contralateral. Thus, flow thresholds for the infarct development were considered to be 12–15 ml 100g. min. (40% of contralateral values) in this model..We successfully elicited SD in 9 m

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