Titlebook: Sodium Channels, Pain, and Analgesia; Michael J. Parnham (Senior Scientific Advisor),Kev Book 2005 Birkh?user Basel 2005 Nervous System.So

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Andreas Scholztrometers was increased by nearly an order of magnitude by the instrument manufacturers. Furthermore, the technique of tandem mass spectrometry (MS/MS) was give978-94-010-5157-6978-94-011-2618-2Series ISSN 1389-2185

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Book 2005The final chapter highlights the functional diversity of sodium channels in part provided by post-transcriptional processing and the insights into sodium channel function that are being provided by tissue specific and inducible gene knock-out technology..

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Sodium channels and nociceptive nerve endings,ry pain. For example, increased expression of both TTX-sensitive and TTX-resistant Na. channels may also contribute to lowering the voltage threshold and changing the firing characteristics of nociceptive nerve endings..Based on current evidence, selective blockade of Na. channel subtypes, in partic

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NaV1.8 as a drug target for pain,is now a feasible drug target with cell lines being developed that allow high-throughput screening of Na.1.8 in a physiological context and screening technology being available to measure sodium channel activity in high-throughput functional assays. However, ultimately only the availability of drugs

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metric analysis can readily be expanded for analysis of other lipid classes not mentioned as long as appropriate sample preparation is conducted, and should aid researchers in the field to better understand and manage the technology for analysis of cellular lipidomes.

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