Titlebook: Side-Effects of Anti-Inflammatory Drugs 3; K. D. Rainsford,G. P. Velo Book 1992 Springer Science+Business Media Dordrecht 1992 kidney.live

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The ulcerogenic and anti-haemostatic effects of NSAIDs in the gut provoke bleeding because of their anti-haemostatic activity [1]. Observations in favour of the latter proposition are that NSAIDs increase the incidence of bleeding but not uncomplicated duodenal ulcer and probably of non-ulcer bleeding; NSAID bleeding often occurs early or with sporadic consumptio

Acetaminophen

What role does , infection play in NSAID-associated gastric inflammation?een proposed. Although the organism only colonizes the epithelial cells of the gastric mucosa, it has been found in ectopic gastric mucosa and more importantly in areas of gastric metaplasia in the duodenal bulb [4], a vital link in the explanation of its role in duodenal mucosal damage. The particu

bibliophile

Endoscopic evaluation of NSAID ulcerationar injury were reported in the medical literature. The advent of fibreoptic endoscopy and photography in the 1960s made it much easier to evaluate the effects of aspirin and other non-steroidal anti-inflammatory agents (NSAIDs) on the gastric and duodenal mucosa. In 1975 we reported the first double

Aqueous-Humor

Intestinal mucosal permeability in inflammatory rheumatic diseases as a selective barrier to the luminal material, consisting not only of nutrients, water and electrolytes but also of bacteria and antigens. Abnormalities in the gut membrane, e.g. inflammation or disturbed permeability, could lead to an enhanced uptake of antigens which can induce inflammatory resp