Titlebook: Side Effects of Anti-Inflammatory Drugs IV; The Proceedings of t K. D. Rainsford Conference proceedings 1997 Springer Science+Business Medi

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Measuring Beneficial and Adverse Events in Osteoarthritis Clinical Trialssult in different event rate scores. The optimum method of assessing beneficial and adverse events in OA clinical trials has yet to be elucidated but is the subject of ongoing research and discussion.

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Oral Tolerance from a General Perspective, and the Possible Role of Side-Effects in the Gut0–20%). It is encouraging to find that those patients who did respond to the highest dosage (10 mg/day for 12 weeks) also showed a small but significant drop in their anti-collagen antibody titre. A possible explanation is that the responders had an appropriate level of gut permeability, perhaps associated with their response to NSAID treatment.

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The Placebo Response: Clinical Efficacy and Toxicitya placebo, it being considered outside proper professional practice. Many physicians consider it an affront of their ‘scientific’ persona. Measurement of placebo effect can only be done by including a group of patients in a clinical therapeutic trial who receive no scientific therapy. Nocebo effects

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Side-Effects of Non-Steroidal Anti-Inflammatory Drugs: Studies from the Tayside Medicines Monitoringlogical research. MEMO links community dispensed prescribing data with hospitalization morbidity data. MEMO collects data on all dispensed drugs in Tayside. Data on all NSAIDs and ulcer healing drugs (UHDs) are available from 1989 onwards. Using this resource, studies that have been carried out have

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Side-Effects from Biologics in the Therapy of Rheumatic Diseasesgainst T cells, cytokines or adhesion molecules, soluble forms of cytokine receptor (e.g. soluble TNFα receptor or soluble IL-1 receptors) and recombinant forms of naturally occurring cytokine antagonists (e.g. IL-1 receptor antagonists). These agents are produced by recombinant DNA technology, and

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