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Titlebook: Shock, Sepsis, and Organ Failure; First Wiggers Bernar Günther Schlag,Heinz Redl,John H. Siegel Conference proceedings 1990 Springer-Verlag

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發(fā)表于 2025-3-21 16:18:50 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Shock, Sepsis, and Organ Failure
副標(biāo)題First Wiggers Bernar
編輯Günther Schlag,Heinz Redl,John H. Siegel
視頻videohttp://file.papertrans.cn/867/866693/866693.mp4
圖書封面Titlebook: Shock, Sepsis, and Organ Failure; First Wiggers Bernar Günther Schlag,Heinz Redl,John H. Siegel Conference proceedings 1990 Springer-Verlag
描述The First Wiggers Bernard Conference on Shock, Sepsis, and Organ Failure was envisaged as a meeting of American and European Scientists. It was named after two researchers, one from the "Old" and one from the "New World", who are wellknown for their outstanding achievements in shock research and experimental medicine. This meeting was a point of de- parture for a series of workshops that will focus on two to three main topics dealt with in detail by experts in their respective fields. The First Wiggers Bernard Conference addressed three main questions: 1. Multi-organ-failure - is it a sequence of organ failures or a systemic disease? 2. Cellular events in trauma and sepsis - are they due to suppression, activation or both? 3. Mediators of organ failure - are LPS or sepsis necessary? Although many answers were given in this first session, numerous issues remain to be clarified due to the enormous complexity of the subject. How- ever, the stage was set for encounters between clinicians and basic medical researchers, since theoretical knowledge will be useful to the clinician ex- ploring new approaches to diagnosis, prophylaxis, and therapy. The conference could not have been organize
出版日期Conference proceedings 1990
關(guān)鍵詞Organversagen; Schock; Sepsis; Shock; organ failure; trauma
版次1
doihttps://doi.org/10.1007/978-3-642-75644-3
isbn_softcover978-3-540-52495-3
isbn_ebook978-3-642-75644-3
copyrightSpringer-Verlag Berlin, Heidelberg 1990
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發(fā)表于 2025-3-21 23:18:35 | 只看該作者
Humoral and Cellular Activation in Blood Leading to Organ FailurePosttraumatic organ damage is related to the effects of a multitude of mediators released via activation of the humoral and blood cell cascade systems. Thus several generations of toxic messengers may be released.
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Posttraumatic Immune Suppression as Initiator of Organ FailureEndotoxin mediates the pathophysiological changes associated with sepsis and multisystem organ failure (MSOF).
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Endothelium as the Interface between Blood and Organ in the Evolution of Organ Failureg blood with its cellular components. To perform their task, the EC are able to form a barrier against molecular and cellular components and thus provide a highly specific permeability (Machovich, 1988).
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The Metabolic Basis of the Septic Multiple Organ Failure Syndrome: Abnormal Energetic Fuel Control, iple organ failure syndrome (MOFS) (76). This review will attempt to conceptually integrate those aspects of this process which lead to this metabolic dysfunction and where possible to suggest some of the metabolic and physiologic loci where therapeutic modalities may be directed.
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發(fā)表于 2025-3-23 02:38:38 | 只看該作者
Conference proceedings 1990enormous complexity of the subject. How- ever, the stage was set for encounters between clinicians and basic medical researchers, since theoretical knowledge will be useful to the clinician ex- ploring new approaches to diagnosis, prophylaxis, and therapy. The conference could not have been organize
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