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Titlebook: Sepsis and Organ Dysfunction; ...from Chaos to Rat Organ Failure Academy,A. E. Baue (Emeritus Profess Conference proceedings 2002 Springer-

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發(fā)表于 2025-3-21 16:25:11 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Sepsis and Organ Dysfunction
副標(biāo)題...from Chaos to Rat
編輯Organ Failure Academy,A. E. Baue (Emeritus Profess
視頻videohttp://file.papertrans.cn/866/865346/865346.mp4
概述The volume presents the most recent results of research in the field of sepsis
圖書(shū)封面Titlebook: Sepsis and Organ Dysfunction; ...from Chaos to Rat Organ Failure Academy,A. E. Baue (Emeritus Profess Conference proceedings 2002 Springer-
描述Even if in the last years more information on sepsis and new treatments has become available, mortality rate is however high. In fact, the complexity of the several biohumoral factors involved in this process is difficult to understand. Another major problem is the consequent organ failure, which grows in a sequential way and in relation with the lesion gravity and the functional reserves of the patient. The volume presents the most recent results of research in this field.
出版日期Conference proceedings 2002
關(guān)鍵詞Immune system; Sepsis; antibiotics; bacteria; bacterial infection; bacterial translation; endotoxin; infect
版次1
doihttps://doi.org/10.1007/978-88-470-2213-3
isbn_softcover978-88-470-0178-7
isbn_ebook978-88-470-2213-3
copyrightSpringer-Verlag Italia, Milano 2002
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Septic Shock TherapySepsis represents one of the pathological conditions that can develop during an intensive care unit (ICU) stay or can be the cause of admission to the ICU. In the past the lack of a commonly accepted definition of this pathology led to a common misinterpretation of morbility and mortality [1–3].
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https://doi.org/10.1007/978-88-470-2213-3Immune system; Sepsis; antibiotics; bacteria; bacterial infection; bacterial translation; endotoxin; infect
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978-88-470-0178-7Springer-Verlag Italia, Milano 2002
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Alveolar Epithelium in Host Defence: Cytokine Productioneobronchitis, bronchiolitis and pneumonia, are significant clinical problems. The increasing number of multidrug-resistant microbes has made the treatment of these infections much more difficult [1]. To further improve therapies for respiratory infection, we need to learn more about the host defence in the lung.
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