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Titlebook: Role of Apoptosis in Infection; Diane E. Griffin Book 2005 The Editor(s) (if applicable) and The Author(s), under exclusive license to Spr

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發(fā)表于 2025-3-21 19:14:17 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Role of Apoptosis in Infection
編輯Diane E. Griffin
視頻videohttp://file.papertrans.cn/832/831513/831513.mp4
叢書名稱Current Topics in Microbiology and Immunology
圖書封面Titlebook: Role of Apoptosis in Infection;  Diane E. Griffin Book 2005 The Editor(s) (if applicable) and The Author(s), under exclusive license to Spr
出版日期Book 2005
關(guān)鍵詞Malaria; apoptosis; cell; immune response; immunity; infection; infections; insects; molecular mechanisms; pa
版次1
doihttps://doi.org/10.1007/b138916
isbn_softcover978-3-642-06192-9
isbn_ebook978-3-540-27320-2Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
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Interactions Between Malaria and Mosquitoes: The Role of Apoptosis in Parasite Establishment and Veite. Apoptosis also occurs in cells of the midgut epithelium that have been invaded by the parasite and are subsequently expelled into the midgut. In addition, the parasite itself dies by a process of programmed cell death (PCD) in the lumen of the midgut before invasion has occurred. Caspase-like a
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Apoptosis and Its Modulation During Infection with ,: Molecular Mechanisms and Role in Pathogenesisere pathology or facilitates a stable parasite-host-interaction. However, . has also evolved strategies to inhibit host cell apoptosis. Apoptosis is blocked by indirect mechanisms in uninfected bystander cells, thereby modulating the inflammatory response to the parasite. In contrast, inhibition of
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Activates Host Cell Caspases During Contact-Dependent Cell Killing,rovided by the Gal/GalNAc adherence lectin. Host cells are lysed in a contact-dependent fashion. There is evidence that suggests that this contact-dependent killing involves the induction of the host cell’s apoptotic machinery. Phagocytosis can then occur, consistent with metazoan apoptotic clearance.
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