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Titlebook: Reviews of Physiology, Biochemistry and Pharmacology, Vol. 167; Bernd Nilius,Thomas Gudermann,Ole H. Petersen Book 2014 The Editor(s) (if

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書目名稱Reviews of Physiology, Biochemistry and Pharmacology, Vol. 167
編輯Bernd Nilius,Thomas Gudermann,Ole H. Petersen
視頻videohttp://file.papertrans.cn/830/829687/829687.mp4
概述Provides high quality, in-depth reviews covering the full range of modern physiology, biochemistry and pharmacology.Aims to filter, highlight and review the latest development in these rapidly advanci
叢書名稱Reviews of Physiology, Biochemistry and Pharmacology
圖書封面Titlebook: Reviews of Physiology, Biochemistry and Pharmacology, Vol. 167;  Bernd Nilius,Thomas Gudermann,Ole H. Petersen Book 2014 The Editor(s) (if
描述The highly successful Reviews of Physiology, Biochemistry and Pharmacology continue to offer high-quality, in-depth reviews covering the full range of modern physiology, biochemistry and pharmacology. Leading researchers are specially invited to provide a complete understanding of the key topics in these archetypal multidisciplinary fields. In a form immediately useful to scientists, this periodical aims to filter, highlight and review the latest developments in these rapidly advancing fields.
出版日期Book 2014
關鍵詞SUDEP; glutamate receptors; nerve injury; neuroglia; neuropathy; ?-amyloid protein
版次1
doihttps://doi.org/10.1007/978-3-319-11921-2
isbn_softcover978-3-319-36407-0
isbn_ebook978-3-319-11921-2Series ISSN 0303-4240 Series E-ISSN 1617-5786
issn_series 0303-4240
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerl
The information of publication is updating

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Reviews of Physiology, Biochemistry and Pharmacology, Vol. 167
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The TRPA1 Channel in Inflammatory and Neuropathic Pain and Migraine,nsition of mechanical and cold hypersensitivity from an acute to a chronic condition and as the primary transducing pathway by which oxidative/nitrative stress produces acute nociception, allodynia, and hyperalgesia. A series of migraine triggers or medicines have been reported to modulate TRPA1 act
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,Glial Calcium Signalling in Alzheimer’s Disease,alling in astrocytes exposed to exogenous β-amyloid or in astrocytes from transgenic Alzheimer disease animals models, characterized by endogenous β-amyloidosis. Accumulated experimental data indicate deregulation of Ca. homeostasis and signalling in astrocytes in AD, which should be given full path
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