Titlebook: Restenosis after Intervention with New Mechanical Devices; Patrick W. Serruys (Professor of Interventional Ca Book 1992 Springer Science+B

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Evaluation of Restenosis Following New Coronary Interventions and technical advancement [1, 2]. If significant restenosis is defined as late luminal narrowing greater than 50%, approximately 30–35% of patients will manifest such restenosis by 3 to 6 months following conventional balloon angioplasty [3–5]. Nearly two dozen “second generation” coronary interven

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Introduction: Stenting for Restenosis?Philip Urban, at that time a research fellow at the University Hospital of Lausanne in 1987, found an article published by Alexis Carrel from New York in September 1912 in the American monthly “Surgery, Gynaecology and Obstretrics” dealing with such ideas [1]. In this article Dr Carrel described the

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The Wallstent Experience: 1986–1990imitations of coronary angioplasty, early acute occlusion and late restenosis [1]. As with all new procedures, operators of the device had to struggle with their own learning curves at the same time that anticoagulation regimens and clinical indications and contraindications evolved from their clini

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Restenosis after Gianturco-Roubin Stent Placement for Acute Closuredural occlusions occurred in 6.8% of patients in the 1985–1986 NHLBI Registry and was associated with a five-fold increase in the incidence of death (5% compared to 1%) and an increase in myocardial infarction incidence from a few percent in those without acute closure to 27% in those who closed and

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Immediate and Long-term Morphologic Changes in Stenosis Geometry after Wiktor? Stent Implantation inobstructive coronary artery disease. In most western countries, the number of PTCA’s equals or even exceeds the number of coronary artery bypass operations [1]. Although most stenoses can now be traversed and dilated by balloon angioplasty with a high initial success rate and a low complication rate

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Atherectomy Introductionsty (PTCA) [1]. Dilatation of stenoses by balloons frequently causes splitting of the atheroma or normal vessel wall while creating a larger lumen. In contrast, atherectomy was designed to create a larger lumen by removing obstructive tissue [2, 3]. The concept of atherectomy is based on the hypothe

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Restenosis: Directional Coronary Atherectomyan obstructed vessel would create a smooth and wide lumen [1, 2]. A smooth, wide lumen without dissection may prevent acute occlusion or reduce thrombus formation, thus improving the acute outcome. Improved flow pattern may prevent platelet aggregation or thrombus formation which are potential trigg

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