Titlebook: Relaxin 2000; Proceedings of the T Geoffrey W. Tregear,Richard Ivell,John D. Wade Conference proceedings 2001 Springer Science+Business Med

Condescending

吝嗇性

Relaxin is an important factor for uterine differentiation and implantation in the marmoset monkey birth canal and at least in some species a suppression of spontaneous contractions [1,2]. Such findings are generally observed in animal models, like the rat and pig, which show a marked relaxin physiology with maximum relaxin levels during the last trimester of pregnancy. For primates, however, th

DAUNT

oblique

promote

Intersection of the relaxin and estrogen signalling pathways in the uteruse uterus during pregnancy, remodelling of the reproductive tract in preparation for parturition, and development of the mammary glands [1]. The mechanisms by which relaxin exerts these effects, however, remain unclear, in large part because the relaxin receptor has not yet been cloned. Interestingly

某人

Relaxin up-regulates rat uterine vascular endothelial growth factor mRNA ,reproductive tract prior to parturition [1]. Most of these well-established effects of relaxin have been characterized with regard to relaxin’s systemic effects as a circulating factor released from the corpus luteum during pregnancy and prior to parturition [2,3]. Many of relaxin’s acute effects, h

BARGE

Comparative aspects of relaxin and relaxin-like factor (Insulin 3) in the placental trophoblastng or fusing with the maternal mucosa. The dominating placenta of most mammals is the chorio-allantoic placenta. Both members of the relaxin family, relaxin itself and the structurally closely related relaxin-like factor (RLF) also known as INSL3 [1, 2], are expressed at the fetomaternal interface o

同步信息

Secretion of matrix metalloproteinase-2, tissue inhibitor of metalloproteinase-1 and human chorionicydig insulin-like peptide (LEY I-L, RLF, INSL3) and peptides encoded by INSL4 (Early placenta insulin-like), INSL5 and INSL6 genes [1-7]. Whereas the physiological role of relaxin has been well assessed in late pregnancy and parturition [8,9], recent studies in primates and humans have shown that re

充滿人

Molecular remodelling of members of the relaxin family during primate evolutiongh sequence variability within and between taxonomic groups among land animals. Widely regarded as a hormone of the female reproductive tract, relaxin is expressed in the ovary and at the feto-maternal interface [1]. Known as a marker of human postpubertal testicular Leydig cells in various species

Presbycusis

Ynes, California in 1981 and then the First International Congress on the Hormone Relaxin in Florence in 1982. After a hiatus of several years, the Second International Congress on the Hormone Relaxin was held in Adelaide in 1994. Since that time, the enormous advances in molecular biology and prot