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Titlebook: Recovery from Brain Damage; Research and Theory Stanley Finger Book 1978 Plenum Press, New York 1978 Deprivation.Syndrom.Training.attention

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書目名稱Recovery from Brain Damage
副標(biāo)題Research and Theory
編輯Stanley Finger
視頻videohttp://file.papertrans.cn/825/824293/824293.mp4
圖書封面Titlebook: Recovery from Brain Damage; Research and Theory Stanley Finger Book 1978 Plenum Press, New York 1978 Deprivation.Syndrom.Training.attention
描述It has long been recognized that damage to the mammalian central nervous system may be followed by behavioral recovery, but only re- cently has close attention been directed to specific factors which may enhance or retard restitution. This is evident in the rapidly growing number of journal articles and scientific paper sessions dealing with "recovery of function," as well as in the publicity given by the popular press to some of the findings in this field. The present text seeks to examine the foundations of brain lesion research, to review recent material on a number of factors which ap- pear to contribute to recovery after brain damage, and to present mod- els which have been proposed to account for these effects. In order to best accomplish these goals, a number of key workers in these areas were asked to examine and describe research literatures dealing with specific problems or methodological manipulations associated with brain damage and behavior, using their own experiments and those of others to illustrate important points. In addition, significant interpre- tive and theoretical issues were to be evaluated in each chapter.
出版日期Book 1978
關(guān)鍵詞Deprivation; Syndrom; Training; attention; behavior; brain; central nervous system; nervous system; neuropsy
版次1
doihttps://doi.org/10.1007/978-1-4615-7723-2
isbn_softcover978-1-4615-7725-6
isbn_ebook978-1-4615-7723-2
copyrightPlenum Press, New York 1978
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ome proliferator-activated alpha nuclear receptor (PPARα), which regulates genes involved in lipid metabolism and inflammatory responses, is a viable target for treating nicotine dependence. The endogenous ligands for PPARα, oleoylethanolamide and palmitoylethanolamide, are structurally similar to a
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ome proliferator-activated alpha nuclear receptor (PPARα), which regulates genes involved in lipid metabolism and inflammatory responses, is a viable target for treating nicotine dependence. The endogenous ligands for PPARα, oleoylethanolamide and palmitoylethanolamide, are structurally similar to a
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Thomas A. Woolsey00 Menschen wurden ab 1934 gegen ihren Willen sterilisiert, mehr als 200.000 Menschen in Heil- und Pflegeanstalten ermordet. Bei der Selektion der Patienten wurde der vermeintliche ?Wert? des Menschen zum leitenden Gesichtspunkt. ?rzte, Pflegende und Funktion?re urteilten nach Ma?gabe von ?Heilbarke
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