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Titlebook: Recombination and Meiosis; Crossing-Over and Di Richard Egel,Dirk-Henner Lankenau Book 2008 Springer-Verlag Berlin Heidelberg 2008 Chromoso

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發(fā)表于 2025-3-21 18:42:52 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Recombination and Meiosis
副標(biāo)題Crossing-Over and Di
編輯Richard Egel,Dirk-Henner Lankenau
視頻videohttp://file.papertrans.cn/825/824111/824111.mp4
概述First book to give a detailed insight in the processes and mechanisms taking place in the cell during meiosis and recombination.Written by high-impact authors from the field
叢書名稱Genome Dynamics and Stability
圖書封面Titlebook: Recombination and Meiosis; Crossing-Over and Di Richard Egel,Dirk-Henner Lankenau Book 2008 Springer-Verlag Berlin Heidelberg 2008 Chromoso
描述.Once per life cycle, mitotic nuclear divisions are replaced by meiosis I and II – reducing chromosome number from the diploid level to a haploid genome, reshuffling the homologous chromosomes by their centromeres, and recombining chromosome arms by crossing-over. In animals, including humans, all this happens during the germ cell formation of eggs and sperm. Due to the reign of meiosis, no child is a true genetic copy of either parent. Central to mainstream meiosis, the mechanisms of reciprocal exchange at crossover/chiasma sites stand out as a controlled program of biologically significant molecular changes. To initiate the meiotic exchange of DNA, surgical cuts are made as a form of calculated damage that is subsequently repaired by homologous recombination. These key events are accompanied by ancillary provisions at the level of chromosome core organization, sister chromatid cohesion, and differential centromere connectivity. Great progress has been made in recent years to further our understanding of these mechanisms. Questions still open primarily concern the placement of and mutual coordination between neighboring crossover events. The current book addresses these processes
出版日期Book 2008
關(guān)鍵詞Chromosom; Chromosome Dysjunction; DNA; DNA Double Strand Breaks; Homolog Synapsis; Meiosis; eukaryote; evo
版次1
doihttps://doi.org/10.1007/978-3-540-75373-5
isbn_softcover978-3-642-09459-0
isbn_ebook978-3-540-75373-5Series ISSN 1861-3373 Series E-ISSN 1861-3381
issn_series 1861-3373
copyrightSpringer-Verlag Berlin Heidelberg 2008
The information of publication is updating

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Meiotic Crossing-Over and Disjunction: Overt and Hidden Layers of Description and Control,ally propagated generations, the regularities of Mendeliangenetics and the segregation of partly recombined chromosomes at meiosis are two complementary faces ofone and the same coin. This chapter opens the first book of two in a?series, both volumes being dedicatedto the complex process of meiotic
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Synaptic and Recombination Nodules in Mammals: Structural Continuity with Shifting Protein Composit different organisms. In many species, electron-densenodules are observed in association with pairing forks and/or fully established synaptonemal complexes.However, there are significant differences in the time of appearance, the relative number of early vs. latenodules, and even in the size and sha
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Human Recombination Hotspots: Before and After the HapMap Project,ch as ..However, several intriguing features of human recombination have been unveiled by new analytical methods,including significant differences in both crossover rate and distribution between individuals and acrosschromosomes. Furthermore, studies at the highest resolution have shown that human m
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,Meiotic Nondisjunction—The Major Cause of Trisomy 21, human trisomy 21 that has accumulated during the last15 years is summarized. The first molecular correlate of nondisjunction in humans is altered recombination,meiosis?I errors being associated with reduced recombination and maternal meiosis?II errors withincreased recombination between the nondisj
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