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Titlebook: Recent Developments in Alcoholism; Volume 2 Marc Galanter,Henri Begleiter,David Theil Book 1984 Plenum Press, New York 1984 Syndrom.attenti

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書目名稱Recent Developments in Alcoholism
副標(biāo)題Volume 2
編輯Marc Galanter,Henri Begleiter,David Theil
視頻videohttp://file.papertrans.cn/824/823126/823126.mp4
圖書封面Titlebook: Recent Developments in Alcoholism; Volume 2 Marc Galanter,Henri Begleiter,David Theil Book 1984 Plenum Press, New York 1984 Syndrom.attenti
描述From the President of the Research Society on Alcoholism The field of alcohol research has been slowly but continuously evolving, taking into its domain an ever-increasing array of scientific disciplines. This senes is designed to fill the need for ,a review publication that covers the broad range of research into alcohol actions and alcoholism. Research in alcohol concerns social, epidemiological, and legal concerns in addition to biomedical and behavioral topics to greater degree than research with many other drugs. A publication devoted to alcohol research should serve the broadest existing research community, but perhaps more important, it should also provide a means to recruit new investigators with fresh approaches to the field. We can and must demonstrate that legitimate, high-quality research is being done, but we must also highlight the opportunity for new workers to make a real impact on the problem. The Research Society on Alcoholism seeks to provide such a service not only through this publication but also through other ac- tivities. Richard A. Deitrich, Ph.D.
出版日期Book 1984
關(guān)鍵詞Syndrom; attention; behavior; identity; intervention; perception; physiology; psychiatry; rehabilitation
版次1
doihttps://doi.org/10.1007/978-1-4684-4661-6
isbn_softcover978-1-4684-4663-0
isbn_ebook978-1-4684-4661-6
copyrightPlenum Press, New York 1984
The information of publication is updating

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https://doi.org/10.1007/978-1-4684-4661-6Syndrom; attention; behavior; identity; intervention; perception; physiology; psychiatry; rehabilitation
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Alcohol-Induced Liver Injuryer lobule from the perfused rat liver. With both techniques, ethanol treatment increased the hepatic oxygen gradient. This increase was blocked by the antithyroid drug propylthiouradl. Thus, these experiments provide evidence in support of the hypothesis that pericentral hypoxia is involved in the m
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Hypermetabolic State and Hypoxic Liver Damage hypermetabolic state, while increased circulating levels of these hormones are not required. Antithyroid drugs inhibit both metabolic tolerance . and the hypermetabolic state. While the hypermetabolic state requires an increased ATP utilization in the form of an adenosine triphosphatase, or an inhi
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Alcohol-Induced Mitochondrial Changes in the Liverxidase, and the ATP synthetase complex. On the other hand, no consistent pattern has emerged as to the effect of chronic ethanol consumption on the composition of the membrane phospholipids. Many of the changes appear to be dependent on the sex of the animal, the dietary status, and the duration of
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