Titlebook: Recent Advances in Neurotraumatology; Norio Nakamura (Chaiman and Professor),Takuo Hashi Book 1993 Springer-Verlag Tokyo 1993 Chronic Subd

使混合

The Role of the Kallikrein-Kinin System in Chronic Subdural Hematomaacrocapillaries, perivascular hemorrhages, migration of all inflammatory cells and interstitial edema in the outer membrane have been suggested as factors which cause hematoma enlargement. Bradykinin (BK), an end product of the kallikrein-kinin system, causes enhancement of vascular permeability, va

inconceivable

transplantation

The Role of Platelet-Activating Factor (PAF) in the Development of Chronic Subdural Hematoma in the hematoma of the old hematoma group(more than 14 days from the day symptoms appeared to the day of operation)was lower than that in the fresh hematoma group(less than 14 days)(p<0.05). On the other hand, the PAF-acetylhydrolase activity in the hematoma of the old hematoma group was higher tha

abject

不足的東西

BARB

Demonstrate

大量殺死

Evaluation of Cerebral Blood Flow by SPECT in Patients with Chronic Subdural Hematomas in cerebral blood flow(CBF), especially in the deep cerebral areas of the basal ganglia and thalamus ipsilateral to the hematoma. Depressed CBF in deep cerebral areas was thought to be the common pathogenesis of CSH..Cerebral diaschisis was closely related with severe dementia or disturbance of con

代理人

Significance of Cholesterol Metabolites in Chronic Subdural Hematomay of a biological detoxication mechanism is discussed. Another possible mechanism for the accumulation of 7α-hydroxy-3-Oxo-4-cholestenoic acid is a concentration of this and other polar metabolites in the hematoma.