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Titlebook: Real-Time Systems Engineering and Applications; Engineering and Appl Michael Schiebe,Saskia Pferrer Book 1992 Springer Science+Business Med

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發(fā)表于 2025-3-28 15:00:38 | 只看該作者
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M. Schiebe supplementary material: Overall, this book illustrates the complexities of the regulation and deregulation of genes mediated through epigenetics in the development and progression of human malignancies. All the articles have been carefully chosen to represent several cancer systems with state of ou
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E. Schniederssion of human malignancies. All the articles have been carefully chosen to represent several cancer systems with state of our knowledge on the role of epigenetic deregulation of microRNAs (miRNAs) and their target mRNAs along with epigenetic deregulation of mRNAs. This book also illustrates the rol
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Peter Hruschkaes. PcG proteins maintain silent states of gene expression, while the trxG of genes counteracts silencing with a chromatin opening function. These factors form multimeric complexes that act on their target chromatin by regulating post-translational modifications of histones as well as ATPdependent r
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Wolfgang A. Halang,Karlotto Mangoldes. PcG proteins maintain silent states of gene expression, while the trxG of genes counteracts silencing with a chromatin opening function. These factors form multimeric complexes that act on their target chromatin by regulating post-translational modifications of histones as well as ATPdependent r
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發(fā)表于 2025-3-29 20:45:25 | 只看該作者
K. F. Gebhardtfactors on inheritance. Epigenetic states may contribute to the penetrance of genetic polymorphisms or mutations and thereby modify inheritance patterns. This may result in non‐Mendelian inheritance of genetic traits such as observed in common human disease. The relationship between epigenetics and
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發(fā)表于 2025-3-30 07:12:55 | 只看該作者
Thomas Bemmerl, exposure to an environment that induces susceptibility to a disease may produce similar familial aggregations when the environment is shared by family members. In general, according to the principles of (Johannsen 1903), the emergence of a disease phenotype is the result of the combined effects of
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