Titlebook: Quantitative Mathematical Models in Radiation Biology; Proceedings of the S Jürgen Kiefer Conference proceedings 1988 Springer-Verlag Berli

Kidney-Failure

暴行

The Role of Energy Distributions of Charged Particles in the Mutagenic Radiation Action,s Escherichia coli owing to the fact that the pathways of DNA repair are well known and that there are many mutants deficient in some repair. Both direct mutations and revertants could be used. Our preliminary results suggest that heavy ions can be both more and less effective in comparison with γ-r

LANCE

Relative Biological Effectiveness: Review of a Model,ial issue of radiation biophysics. For example, the early target theory had remained a rather abstract and ambiguous scheme until D.E. Lea in his noted work “Actions of Radiations on Living Cells” [1] drew the conclusions, quantitatively, from simple target models regarding the influence of radiatio

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Hit-Size Effectiveness Approach in Biophysical Modeling,s biological end points in the different biological systems. However, when considering biophysical modeling, it is essential to classify these models into two basic categories: (a) mechanistic models and (b) phenomenological of empirical. With mechanistic models a certain mode of interaction of radi

怪物

Interpreting Survival Observations Using Phenomenological Models,ochemical processes. However, these processes have not been adequately described to allow predictions of the effects at low doses or of radiations having higher stopping powers. The kinetics of damage induction and removal have been studied extensively, even though specific mechanisms have yet to be

GRACE

Cluster Theory of the Effects of Ionizing Radiations,s chromatin structures. The multiple yield of such lesions depends on the radial distribution of deposited energy and on the microstructure of the distribution of DNA in the cell nucleus, both of which place an upper limit on the number of lesions that can be produced by a single particle. The numbe

GUISE

Fantasy

IVORY

A Repair Fixation Model Based on Classical Enzyme Kinetics, take into account. There is a considerable number of suggestions in the literature (see Weber, this volume). Many of them have in common that damage and repair are treated differently. It is, e.g., often assumed that permanent lesions are formed by pairwiseinteraction of sublesions. It is the aim o

Hectic