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Titlebook: Quantitative EPR; Gareth R. Eaton,Sandra S. Eaton,Ralph T. Weber Book 2010 Springer-Verlag Vienna 2010 EPR.Quantitative EPR.calibration.di

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41#
發(fā)表于 2025-3-28 16:06:32 | 只看該作者
42#
發(fā)表于 2025-3-28 22:28:54 | 只看該作者
Implanted widegap II–VI materials for electro-optic applications and electron-beam-pumped devicesuctures and behaviours of defects and impurities. In this chapter, several approaches for ion implantation into widegap II–VI materials, especially ZnS, ZnSe CdS, are reviewed from the points of view of understanding defects and emission centres, and realizing p-n junction formation.
43#
發(fā)表于 2025-3-29 02:28:54 | 只看該作者
https://doi.org/10.1057/9780230304017ne or in combination with other therapeutic regimens, will contribute to halt tumor progression by counteracting tumor-immune escape. Here we describe a selection of methods used to investigate the role of galectin-1 in tumor-immune escape.
44#
發(fā)表于 2025-3-29 03:04:47 | 只看該作者
Nobuaki Ishii,Yuichi Takano,Masaaki Murakist researchers, in my realm of experience, to pay close attention to ethical concerns, and I am greatly appreciative of his contribution to both my professional growth and the development of various scenarios discussed in this text. The portions of this text dealing with confidentiality are the result of many978-1-4757-8558-6978-0-306-46800-1
45#
發(fā)表于 2025-3-29 09:52:15 | 只看該作者
Sickle Cell Disease and Acute Chest Syndrome: Mechanisms and Pathogenenesisthat the lung injury in ACS is caused by acute inflammation directly involving extracellular heme, a prototypical erythroid DAMP molecule released by hemolysis. In support of this model, the infusion of purified heme triggers a lethal acute lung injury in transgenic SCD mice that exhibit many of the
46#
發(fā)表于 2025-3-29 14:19:43 | 只看該作者
47#
發(fā)表于 2025-3-29 17:08:50 | 只看該作者
48#
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發(fā)表于 2025-3-30 02:07:32 | 只看該作者
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