Titlebook: Parathyroid Glands in Chronic Kidney Disease; Adrian Covic,David Goldsmith,Pablo A. Ure?a Torres Book 2020 Springer Nature Switzerland AG

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Effect of PTH on the Hematologic System,ute in close proximity to the endosteal surfaces, form specific microenvironment, called niche, and achieve self-renewal and differentiation, communicating with cells involved in bone metabolism. PTH plays important roles not only in differentiation of osteoblastic lineage but also in blood cell pro

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Control of Secondary Hyperparathyroidism (SPHT) by Older and Newer Vitamin D Compounds,asis of calcium, phosphorus, and vitamin D metabolism caused by declining renal function in chronic kidney disease. This abnormal secondary hyperparathyroidism leads to cardiovascular and extravascular calcification and is directly linked to an increased risk of cardiovascular morbidity and mortalit

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The Role of the Old and the New Calcimimetic Agents in Chronic Kidney Disease-Mineral and Bone Disorder,use mortality. The classical treatment for SHPT includes active vitamin D compounds and phosphate binders. However, achieving the optimal laboratory targets is often difficult because vitamin D sterols suppress PTH secretion, but also promote calcium and phosphate intestinal absorption. Calcimimetic

Alveolar-Bone

Parathyroidectomy in Chronic Kidney Disease,dary hyperparathyroidism (SHPT). SHPT includes not only divalent ions derangements, but also renal bone disease (high turnover, low turnover and/or osteomalacia) and accelerated vascular and ectopic calcifications. In this way, SHPT resembles a true clinical syndrome named CKD-MBD, which is a recogn

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