Titlebook: Oxygen Transport to Tissue XXX; Per Liss,Peter Hansell,David K. Harrison Conference proceedings 2009 The Editor(s) (if applicable) and The

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書目名稱Oxygen Transport to Tissue XXX影響因子(影響力)




書目名稱Oxygen Transport to Tissue XXX影響因子(影響力)學(xué)科排名




書目名稱Oxygen Transport to Tissue XXX網(wǎng)絡(luò)公開度




書目名稱Oxygen Transport to Tissue XXX網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Oxygen Transport to Tissue XXX被引頻次




書目名稱Oxygen Transport to Tissue XXX被引頻次學(xué)科排名




書目名稱Oxygen Transport to Tissue XXX年度引用




書目名稱Oxygen Transport to Tissue XXX年度引用學(xué)科排名




書目名稱Oxygen Transport to Tissue XXX讀者反饋




書目名稱Oxygen Transport to Tissue XXX讀者反饋學(xué)科排名

jet-lag

0065-2598 ernational Society on Oxygen Transport to Tissue (ISOTT) was founded. Since then there has been an annual ISOTT meeting. After 24 years of international ISOTT meetings it was decided, at the 2005 summit in Bary, Italy, that the 2007 meeting was to be held in Uppsala, Sweden. Thus, after the Louisvil

細菌等

The Influence Of Flow Redistribution On Working Rat Muscle Oxygenation,kPa) had little effect. When overall flow was decreased by 20%, the anoxic tissue increased to 7.6% and the mean tissue PO2 decreased to 3.22 kPa. The conclusion from these model calculations is, that flow redistribution has little impact on skeletal muscle oxygenation, which is in line with earlier findings for rat heart.

Exploit

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Mini Sensing Chip for Point-of-Care Acute Myocardial Infarction Diagnosis Utilizing Micro-Electro-Muser-friendly assay. The resulting system is a point-of-care, automatic four cardiac marker sensing system with a 2 x 2.5cm sensing chip. An assay requires a 200 μ$L plasma sample and 15-minute assay time.

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Arthropathy

Mathematical Modeling of The Interaction Between Oxygen, Nitric Oxide And Superoxide,, nitric oxide (NO), superoxide (O2·-), peroxynitrite (ONOO-), nitrite (NO2 -) and nitrate (NO3 -) in cylindrical geometry with blood flow through a 30 ?m diameter arteriole. Steady state concentration gradients of all chemical species were predicted for different O2·- production rates, superoxide d

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Haemoglobin Saturation Controls The Red Blood Cell Mediated Hypoxic Vasorelaxation,rom ischemic events. The identity of the vasorelaxant released from RBCs has yet to be elucidated, however growing evidence suggests that nitric oxide bound to the ?93 cysteine residue of haemoglobin (SNO-Hb) may be responsible. The vasorelaxant moiety is released during the transition of haemoglobi

CBC471

Blood Vessel Specific Vaso-Activity To Nitrite Under Normoxic And Hypoxic Conditions, vena cava (IVC) and pulmonary artery (PA)) in a haemoglobin independent/variable oxygen environment. In 95% oxygen constriction to 8g (Ao), 6g (PA) and 4g (IVC) was achieved using 1μM phenylephrine. The same constriction in 1% oxygen required 3μM. During 95% oxygen constriction was consistent and s

occurrence