Titlebook: Oxygen Transport to Tissue XXIV; Jeffrey F. Dunn,Harold M. Swartz Book 2003 The Editor(s) (if applicable) and The Author(s), under exclusi

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Protein C Separation from Human Blood Plasma Derivatives Using Low Cost Chromatographyreatment can cause catastrophic bleeding and other major medical problems. PC treatment has no bleeding or skin necrosis problems because it circulates in the blood as a zymogen and is only activated when and where it is needed..The vitamin K dependent (VKD) proteins are homologous proteins, making

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壓倒性勝利

Oxygenation and Vascular Perfusion in Spontaneous and Transplanted Tumor Modelsurrent studies were undertaken to compare transplantable, murine KHT tumors with primary and 1. generation transplants of spontaneous mammary carcinomas. To evaluate changes in tumor vascular structure and function, immunostaining of total and perfused vascular spacing, and cryospectrophotometric me

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Effect of Mild Hyperglycemia ± Meta-Iodo-Benzylguanidine on the Radiation Response of R3230 Ac Tumorctrode chamber. For . studies, rats bearing R3230 Ac rat mammary adenocarcinomas were utilized. To evaluate changes in tumor oxygenation induced by glucose or MIBG, intratumoral pO. and skeletal muscle pO. were measured using Eppendorf Histography. To find the effect of mild hyperglycemia (i.p., 1 g

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Effect of Onconase ± Tamoxifen on ASPC-1 Human Pancreatic Tumors in Nude Mice in legs of nude mice bearing AsPC-1 human pancreatic carcinoma. Tumor interstitial fluid pressure (TIFP) was measured using the wick-inneedle technique, while intratumoral pO. utilized O. sensitive needle electrodes. TBF was significantly increased by an i.p. injection of ONC, then returned to the

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Oxygenation in a Human Tumor Xenograft: Manipulation Through Respiratory Challenge and Antibody-Direended the application to human tumors implanted in immunocompromised (SCID) mice. This has allowed us both to investigate dynamic response to respiratory challenge (carbogen) and to probe the mechanisms of a new anti-vascular therapy designed to produce tumor-specific infarction.

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