Titlebook: Oxygen Transport to Tissue XVIII; Edwin M. Nemoto,Joseph C. LaManna,David F. Wilson Book 1997 The Editor(s) (if applicable) and The Author

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Histological Assessment of Rodent CNS Tissues to EPR Oximetry Probe Materialwell localized to the point of injection and created mild acute tissue reaction on implantation (which appeared to resolve quickly) and virtually no tissue reaction at later times. The majority of the implanted fusinite and LiPc material was present extracellularly in the brain and spinal cord. MRI

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Activation of Tyrosine Hydroxylase in Striatum of Newborn Piglets in Response to Hypocapnic Ischemiahowed that, after 20 min of recovery, there was an increase in Vmax with no change in Km for pteridine cofactor, compared to control. This is consistant with the enzyme having been covalently modified (activated) during tissue ischemia caused by hyperventilation and remaining activated well into the

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Coronary Flow Response After Myocardial Ischemia May Predict Level of Functional Recoveryd myocardial management, in which the surgeon adapts the cardioprotective strategy to the needs of each patient, rather than adopting an adversarial position. Buckberg (1994) also observes that new cardioprotective techniques are being introduced without an adequate scientific basis.

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The S Factor—A New Derived Hemodynamic Oxygenation Parameter—A Useful Tool for Simplified Mathematicnto equation 1 and solving for Ca — Cv. . Equation 6 can be simplified to: .A previously defined relationship. between mixed venous PO. (PvO.) and. (where calculated P. is 26.6 ± 1.0) can be used to modify S in a clinically relevant manner..The relationship between S and PvO. can be defined by subst

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