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Titlebook: Oxidative Stress in Skeletal Muscle; A. Z. Reznick (Senior Lecturer and Researcher),L. Book 1998 Springer Basel AG 1998 Lipid.Oxidation.a

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樓主: papyrus
21#
發(fā)表于 2025-3-25 06:48:04 | 只看該作者
Vitamin E and its effect on skeletal muscle,cle fibre type distribution, promote deposition of lipid granules, and result in other increased indications of oxidative stress. Oxygen consumption increases markedly during strenuous exercise and is believed to contribute to the increase in oxygen radical formation during exercise. Acute exercise
22#
發(fā)表于 2025-3-25 10:44:25 | 只看該作者
Differential susceptibility of skeletal muscle proteins to free radical-induced oxidative damage ,, or O. increases susceptibility to subsequent generalized proteolysis by endogenous cytoplasmic or lysosomal enzymes. These data support the concept that oxidatively damaged proteins are degraded by specific proteolytic enzyme types (e.g. macropain), rather than by more generalized proteolytic actio
23#
發(fā)表于 2025-3-25 13:58:58 | 只看該作者
Oxidative stress and Ca2+ transport in skeletal and cardiac sarcoplasmic reticulum,ulatory complex. As Ca. rises, Ca. ions bind to the regulatory binding site of the troponin C subunit, thereby activating cross-bridge cyclic and tension development. In striated muscle, the regulation of Ca. is controlled by a combination of influx and efflux processes. The firing of an action pote
24#
發(fā)表于 2025-3-25 16:11:22 | 只看該作者
25#
發(fā)表于 2025-3-25 23:41:53 | 只看該作者
Oxidative damage after ischemia/reperfusion in skeletal muscle,ans point to the involvement of reactive oxygen metabolites (ROS), superoxide, hydrogen peroxide and hydroxyl radicals in the etiology of ischemia-reperfusion damages ROS may be generated during reperfusion by three main metabolic pathways: 1) oxidation of xanthine and hypoxanthine by xanthine oxida
26#
發(fā)表于 2025-3-26 03:27:30 | 只看該作者
27#
發(fā)表于 2025-3-26 07:56:54 | 只看該作者
Oxidative stress and muscle wasting of cachexia,cle, leading to decreased myosin creatinine phosphokinase (MCK) expression and binding activities. The impaired MCK-E box binding activities resulted from abnormal myogenin/Jun-D complexes, and were normalized by the addition of Jun-D, DTT or Ref-1, a nuclear redox protein. Treatment of skeletal mus
28#
發(fā)表于 2025-3-26 11:55:05 | 只看該作者
29#
發(fā)表于 2025-3-26 13:17:31 | 只看該作者
O. H?nninen,M. Atalayriterien zur Quantifi zierung der Leistungsf?higkeit des Sichtprüfrozesses eingeführt. Um Automatisierungsl?sungen in diesem Bereich zu bewerten und sie mit manuell-visuellen Prüf vorg?ngen vergleichen zu k?nnen, besteht daher die Notwendigkeit, geeignete Bewertungskriterien sowie eine verst?ndliche
30#
發(fā)表于 2025-3-26 16:55:36 | 只看該作者
A. Z. Reznick,L. Packer,C. K. Senriterien zur Quantifi zierung der Leistungsf?higkeit des Sichtprüfrozesses eingeführt. Um Automatisierungsl?sungen in diesem Bereich zu bewerten und sie mit manuell-visuellen Prüf vorg?ngen vergleichen zu k?nnen, besteht daher die Notwendigkeit, geeignete Bewertungskriterien sowie eine verst?ndliche
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