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Titlebook: Oxidative Stress and Vascular Disease; John F. Keaney (Associate Professor of Medicine an Book 2000 Springer Science+Business Media New Yo

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書目名稱Oxidative Stress and Vascular Disease
編輯John F. Keaney (Associate Professor of Medicine an
視頻videohttp://file.papertrans.cn/706/705319/705319.mp4
叢書名稱Developments in Cardiovascular Medicine
圖書封面Titlebook: Oxidative Stress and Vascular Disease;  John F. Keaney (Associate Professor of Medicine an Book 2000 Springer Science+Business Media New Yo
描述One of the major biomedical triumphs of the post-World War II era was the defmitive demonstration that hypercholesterolemia is a key causative factor in atherosclerosis; that hypercholesterolemia can be effectively treated; and that treatment significantly reduces not only coronary disease mortality but also all- cause mortality. Treatment to lower plasma levels of cholesterol - primarily low density lipoprotein (LDL) cholesterol - is now accepted as best medical practice and both physicians and patients are being educated to take aggressive measures to lower LDL. We can confidently look forward to important decreases in the toll of coronary artery disease over the coming decades. However, there is still uncertainty as to the exact mechanisms by which elevated plasma cholesterol and LDL levels initiate and favor the progression of lesions. There is general consensus that one of the earliest responses to hypercholesterolemia is the adhesion of monocytes to aortic endothelial cells followed by their penetration into the subendothelial space, where they differentiate into macrophages. These cells, and also medial smooth muscle cells that have migrated into the subendothelial space, th
出版日期Book 2000
關鍵詞atherosclerosis; cardiovascular; gene; gene expression; hypertension; oxidative stress; physiology; signal
版次1
doihttps://doi.org/10.1007/978-1-4615-4649-8
isbn_softcover978-1-4613-7103-8
isbn_ebook978-1-4615-4649-8Series ISSN 0166-9842
issn_series 0166-9842
copyrightSpringer Science+Business Media New York 2000
The information of publication is updating

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Developments in Cardiovascular Medicinehttp://image.papertrans.cn/o/image/705319.jpg
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https://doi.org/10.1007/978-1-4615-4649-8atherosclerosis; cardiovascular; gene; gene expression; hypertension; oxidative stress; physiology; signal
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Jay W. Heinecketic, prolymphoblastic and immunoblastic acute lymphoid leukaemias. Using computer analysis, we demonstrated that, among all the prognostic factors, i.e. type, age, tumour volume, leukocytosis..., the cytological type remains the only one which determines the other factors. The best therapeutic respo
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