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Titlebook: Oxidative Stress and Neuroprotection; H. Parvez,P. Riederer Book 2006 Springer-Verlag Vienna 2006 Alzheimer.Morbus Parkinson.Parkinson.alz

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發(fā)表于 2025-3-21 17:44:56 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Oxidative Stress and Neuroprotection
編輯H. Parvez,P. Riederer
視頻videohttp://file.papertrans.cn/706/705314/705314.mp4
叢書名稱Journal of Neural Transmission. Supplementa
圖書封面Titlebook: Oxidative Stress and Neuroprotection;  H. Parvez,P. Riederer Book 2006 Springer-Verlag Vienna 2006 Alzheimer.Morbus Parkinson.Parkinson.alz
描述.This book deals with basic and clinical aspects of monoamine oxidase (MAO) subtypes A and B highlighting its importance in neurological and psychiatric diseases. Consequently the therapeutic actions of MAO-A and -B inhibitors in Parkinson’s disease (PK) and depression are the focus of several chapters. As MAO is the basis of the development of the "oxidative stress hypothesis" of PD, several chapters are devoted to iron and iron-induced oxidative stress in various experimental studies and clinical conditions. Based on these findings, new compounds have been developed which not only block MAO, but are in addition, either inhibitors of acetylcholine esterase or have iron chelating properties. The aspect of "preclinical" and "clinical" neuro protection as well as MAO neuroprotection are additional topics covered in this book. MAO, iron and neuroprotection are seen in the framework of general anti Parkinson’s therapy with chapters on levodopa, dopaminergic receptor agonists and clinical issues. .
出版日期Book 2006
關(guān)鍵詞Alzheimer; Morbus Parkinson; Parkinson; alzheimer‘s disease; apoptosis; brain; cell; cytokine; dementia; depr
版次1
doihttps://doi.org/10.1007/978-3-211-33328-0
isbn_softcover978-3-7091-1736-1
isbn_ebook978-3-211-33328-0Series ISSN 0303-6995
issn_series 0303-6995
copyrightSpringer-Verlag Vienna 2006
The information of publication is updating

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Acute and chronic effects of developmental iron deficiency on mRNA expression patterns in the braindevelopment in a metabolically compromised setting that given appropriate intervention is mostly correctable. There are, however, long term consequences to the developmental iron deficiency that could underlie the neurological deficits reported for iron deficiency.
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0303-6995 psychiatric diseases. Consequently the therapeutic actions of MAO-A and -B inhibitors in Parkinson’s disease (PK) and depression are the focus of several chapters. As MAO is the basis of the development of the "oxidative stress hypothesis" of PD, several chapters are devoted to iron and iron-induce
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,The DONPAD-study — Treatment of dementia in patients with Parkinson’s disease with donepezil,dizziness and confusion. This may result from the titration regime of donepezil, that allows only 5 and 10mg dosages. Participants with premature study termination had a significant longer duration of PD, less motivation and sleep disturbances at night. Treatment with donepezil was only effective in
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