Titlebook: Occupational Asthma; Torben Sigsgaard,Dick Heederik Book 2010 Birkh?user Basel 2010 Occupational disease.Pulmonary disease.Respiratory Inf

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Natural history and prognosis,orsening with increasing age. Likewise, persistent nonspecific bronchial hyperresponsiveness at long-term follow-up is found in about three quarters of these patients. The situation is even worse if patients with occupational asthma are left in the same workplace. These clinical and functional data

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Cytokines

Mechanisms of occupational asthma caused by low-molecular-weight chemicals,se. The study of the sensitizing and asthma-inducing properties of low-molecular-weight (LMW) agents is evolving quickly. So far, experimental research has shown that OA caused by sensitization to LMW agents does not completely fit the pathways of the traditional allergic model, in which there is a

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Asthma-like diseases in agriculture,arns, and crop farming together with exposure to endotoxin and quaternary ammonium have been described as environmental risk factors for self-reported asthma and/or wheeze in cross-sectional studies. The prevalence of selfreported asthma has been found to range from 0.7% in female greenhouse workers

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Management of an individual worker with occupational asthma,, and to prevent other workers from developing occupational asthma. To do this a confident diagnosis must be made. Occupational asthma should be suspected when asthma symptoms improve on days away from work or on holiday. As these questions have many false-positive answers, the diagnosis needs objec

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