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Titlebook: Obesity; Pathogenesis, Diagno Paolo Sbraccia,Nicholas Finer Living reference work 20200th edition Neuroendocrinology of energy balance.Adi

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發(fā)表于 2025-3-21 19:07:50 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Obesity
副標(biāo)題Pathogenesis, Diagno
編輯Paolo Sbraccia,Nicholas Finer
視頻videohttp://file.papertrans.cn/701/700104/700104.mp4
概述Provides readers comprehensive information on the causes, complications and treatments of obesity.Written by international experts in the field.Covers the full range of pharmacological and surgical tr
叢書名稱Endocrinology
圖書封面Titlebook: Obesity; Pathogenesis, Diagno Paolo Sbraccia,Nicholas Finer Living reference work 20200th edition  Neuroendocrinology of energy balance.Adi
描述.This book presents the latest advances in our understanding of the pathophysiologic basis that induces the chronic positive energy balance necessary to progressively store excessive amounts of fat mass. It highlights the clinical complications of obesity, while also focusing on the major scientific advances that have highlighted the role of genetics, adipose tissue endocrinology and the environment in the mechanisms and causes of obesity.?Particular attention is given to the consequences of obesity, including an ever -increasing number of other diseases such as type 2 diabetes, cardiovascular diseases and cancer, as well as conditions such as arthropathy, asthma, non-alcoholic liver disease, and dementia. Increasingly recognized uniting causes highlight the role of systemic, local and paracrine actions of adipocytokines in causing inflammation.?As we come to better understand how hypothalamic control of energy balance is influenced by endocrine signaling from the gut and adipose tissue, it paves the way for new therapeutic targets and a resurgence of pharmacological and surgical approaches to treatment.?.Written by respected experts in the field, this volume is intended as a major
出版日期Living reference work 20200th edition
關(guān)鍵詞Neuroendocrinology of energy balance; Adipose organ; Eating disorders; Dietary treatment; Pharmacologica
doihttps://doi.org/10.1007/978-3-319-47685-8
isbn_ebook978-3-319-47685-8
issn_series 2510-1927
The information of publication is updating

書目名稱Obesity影響因子(影響力)




書目名稱Obesity影響因子(影響力)學(xué)科排名




書目名稱Obesity網(wǎng)絡(luò)公開度




書目名稱Obesity網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Obesity被引頻次




書目名稱Obesity被引頻次學(xué)科排名




書目名稱Obesity年度引用




書目名稱Obesity年度引用學(xué)科排名




書目名稱Obesity讀者反饋




書目名稱Obesity讀者反饋學(xué)科排名




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The Adipose Organ,-secreting glandular cells (pink adipocytes). During chronic positive energy balance, the adipose organ undergoes a whitening phenomenon with hypertrophic adipocytes. Hypertrophic adipocytes show several alterations of their organelles including those able to activate the inflammasome system. Stress
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Roles of Gut Hormones in the Regulation of Food Intake and Body Weight,earned associations play a relevant role in the anticipation of food reward and pleasure..We report an overview of the intestinal mechanisms regulating satiety and body weight with particular emphasis to the effects of drugs and bariatric/metabolic surgery on gut hormonal secretion.
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Eating Disorders,rgical interventions for obesity require an accurate assessment of eating behavior before, during, and after treatment. This chapter describes the clinical features of the eight DSM-5 diagnostic categories of the FED grouping. Then it looks at binge eating disorder and many other eating patterns tha
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Obesity and NAFLD,ediate hepatic injury, inflammation, and apoptosis. In order to manage obesity and NAFLD, nutritionally based body weight management is the principal modifiable risk factor. Thus, dietary interventions for body weight loss should be the main therapeutic approach, although pharmacotherapy and, in som
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Impact of Obesity on Cardiovascular Disease, than in those with normal weight. Such observations are likely partially driven by reverse causality whereby disease-specific issues drive weight loss rather than higher weight per se being protective..While obesity is most commonly defined by BMI, the importance of body fat distribution and marker
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