Titlebook: OPLL; Ossification of the Atsushi Okawa,Morio Matsumoto,Yoshiharu Kawaguchi Book 2020Latest edition Springer Nature Singapore Pte Ltd. 202

分期

書目名稱OPLL影響因子(影響力)




書目名稱OPLL影響因子(影響力)學科排名




書目名稱OPLL網(wǎng)絡公開度




書目名稱OPLL網(wǎng)絡公開度學科排名




書目名稱OPLL被引頻次




書目名稱OPLL被引頻次學科排名




書目名稱OPLL年度引用




書目名稱OPLL年度引用學科排名




書目名稱OPLL讀者反饋




書目名稱OPLL讀者反饋學科排名

Restenosis

Infantry

History of Researchrior and posterior longitudinal ligaments over 100?years after OPLL was first described by Key. Most of these cases were ossification of the anterior longitudinal ligament. He did not recognize the clinical significance of OPLL.

我不明白

Book 2020Latest editionion of the ligamentum flavum (OLF) and diffuse idiopathic spinal hyperostosis. As such, it offers a valuable resource for all those who want to gain a deeper understanding of OPLL/OLF and those involved in making decisions about its treatment..

Bmd955

essing ossification of the ligamentum flavum (OLF) and diffuse idiopathic spinal hyperostosis. As such, it offers a valuable resource for all those who want to gain a deeper understanding of OPLL/OLF and those involved in making decisions about its treatment..978-981-15-3857-5978-981-15-3855-1

strain

GROSS

Ceramic

spinal ligaments.This book brings together information on all aspects of ossification of the posterior longitudinal ligament (OPLL), a hyperostotic condition of the spine that can cause severe neurological deficits. Since the second edition of?this book was published in 2006, there have been tremend

cultivated

Overview of Possible Roles of OPLL-Associated Genes in OPLL DevelopmentO2 may suppress the differentiation of mesenchymal stem cells toward chondrocytes via the inhibition of canonical Wnt signaling. To obtain a deeper understanding of the pathophysiology of OPLL, appropriate experimental animal models and expression analyses of candidate genes in surgical specimens from OPLL patients are necessary.

minimal

Mechanisms Underlying OPLL Development in Tiptoe Walking (ttw) MiceOPLL model. Ttw mice were subsequently shown to harbor a loss of function mutation in the . (.) gene. Recently, mechanisms underlying ectopic ossifications due to Enpp1 dysfunction have been clarified and shown to involve excessive vitamin D signaling.