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Titlebook: Nutrition and Biotechnology in Heart Disease and Cancer; John B. Longenecker,David Kritchevsky,Marc K. Drez Book 1995 The Editor(s) (if ap

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樓主: 揭發(fā)
11#
發(fā)表于 2025-3-23 10:27:39 | 只看該作者
Jan L. Breslow,Marilyn Dammermanists or scientists from other disciplines, who may use this book as a stimulating starting point for further discoveries and/ or translational endeavors...?.978-3-031-62733-0978-3-031-62731-6Series ISSN 0065-2598 Series E-ISSN 2214-8019
12#
發(fā)表于 2025-3-23 14:21:13 | 只看該作者
13#
發(fā)表于 2025-3-23 18:36:11 | 只看該作者
Jawed Fareed,Debra Hoppensteadt,Lalitha Iyer,Michael Koza,Jeanine M. Walenga,Edward Bermes Jr.terozygosity, to dominant congenital dyserythropoietic anemia type IV invariably caused by a de novo variant in a highly conserved amino acid in the KLF1 DNA-binding domain. In this chapter, we present an overview of the past and present of KLF1 research and discuss the significance of human . variants.
14#
發(fā)表于 2025-3-23 23:25:10 | 只看該作者
Judith K. Christmanterozygosity, to dominant congenital dyserythropoietic anemia type IV invariably caused by a de novo variant in a highly conserved amino acid in the KLF1 DNA-binding domain. In this chapter, we present an overview of the past and present of KLF1 research and discuss the significance of human . variants.
15#
發(fā)表于 2025-3-24 04:18:37 | 只看該作者
16#
發(fā)表于 2025-3-24 08:33:47 | 只看該作者
Thomas J. Slagation factors; E2Fl, E2F2, E2F3, E2F4, or E2F5 can heterodimerize with either DPl or DP2. Cellular promoters whose E2F sites mediate a link between transcription978-3-642-79912-9978-3-642-79910-5Series ISSN 0070-217X Series E-ISSN 2196-9965
17#
發(fā)表于 2025-3-24 11:11:00 | 只看該作者
0065-2598 ers of this conference thought the time was propitious for bringing together knowledge on newer aspects of molecular biological research with current advances i978-1-4613-5804-6978-1-4615-1957-7Series ISSN 0065-2598 Series E-ISSN 2214-8019
18#
發(fā)表于 2025-3-24 18:07:13 | 只看該作者
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發(fā)表于 2025-3-24 19:04:59 | 只看該作者
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發(fā)表于 2025-3-25 01:37:41 | 只看該作者
John R. Guytonmutation and deregulation in cancer cells and TCF family members possess qualities of tumor suppressors as well as tumor promoters. And yet, despite all the progress made, the full range of the functional diversity and the regulatory complexity of TCF proteins is just beginning to emerge.
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