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Titlebook: Non-Alcoholic Steatohepatitis; Methods and Protocol Devanand Sarkar Book 2022 The Editor(s) (if applicable) and The Author(s), under exclus

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樓主: monster
31#
發(fā)表于 2025-3-26 23:28:25 | 只看該作者
32#
發(fā)表于 2025-3-27 01:49:54 | 只看該作者
Analysis of Liver Responses to Non-alcoholic Steatohepatitis by mRNA-Sequencing,hough NASH is associated with excessive liver lipid accumulation, hepatocyte injury, inflammation, and fibrosis, its etiology remains incompletely understood. These can be characterized by determining transcriptional changes in specific genes previously found to be involved in these processes. As an
33#
發(fā)表于 2025-3-27 08:05:02 | 只看該作者
Single Cell RNA Sequencing in NASH,ohepatitis (NASH), scRNA-seq is particularly powerful to understand non-parenchymal cell heterogeneity in the liver, e.g. for inflammatory cells. Myeloid immune cells, particularly macrophages, play a critical role in response of the innate immune system and significantly contribute to the progressi
34#
發(fā)表于 2025-3-27 09:31:15 | 只看該作者
Computational Pipeline for Next-Generation Sequencing (NGS) Studies in Genetics of NASH,fatty liver disease (NAFLD). Genome-wide association studies, whole exome sequencing, and sequencing of entire genes are used to identify variants and/or mutations that predispose to the disease pathogenesis. Here, we present a tutorial that may guide readers to manage high volume of genetics data i
35#
發(fā)表于 2025-3-27 14:33:36 | 只看該作者
Metabolic Phenotyping in Mice with NASH Using Indirect Calorimetry,etes, non-alcoholic steatohepatitis (NASH), cardiovascular diseases, and cancer. Metabolic phenotyping is a set of techniques for studying metabolic dysfunction and behavior information including energy expenditure, body weight gain, glucose homeostasis, and lipid profile. Among different metabolic
36#
發(fā)表于 2025-3-27 17:58:12 | 只看該作者
37#
發(fā)表于 2025-3-27 23:32:23 | 只看該作者
Assessment of Lipotoxic Endoplasmic Reticulum (ER) Stress in Nonalcoholic Steatohepatitis (NASH),asmic reticulum (ER) stress. Consequently, the unfolded protein response (UPR) is initiated, driven by three key ER transmembrane proteins, resulting in downstream responses that are dynamic and interconnected. Thus, careful interrogation of these pathways is required to investigate the complex role
38#
發(fā)表于 2025-3-28 02:57:15 | 只看該作者
Methods in Molecular Biologyhttp://image.papertrans.cn/n/image/666843.jpg
39#
發(fā)表于 2025-3-28 07:08:08 | 只看該作者
https://doi.org/10.1007/978-1-0716-2128-8CRISPR; Kupffer cells; Inflammasome; cell death; NASH
40#
發(fā)表于 2025-3-28 11:29:40 | 只看該作者
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