Titlebook: Nitric Oxide and Inflammation; Daniela Salvemini,Timothy R. Billiar,Yoram Vodovot Book 2001 Birkh?user Verlag 2001 Arthritis.inflammation.

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Wolfram E. Samlowskiitor refresh rate. Performance was assessed with ITR and accuracy and user friendliness was evaluated using a questionnaire. High flickering speed is usually accompanied by poorer BCI performance. In this study, the system utilizing the 200?Hz refresh rate surprisingly competed well in terms of ITR

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Small-Intestine

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Nitric oxide regulation of eicosanoid production,tes. An important link between the NOS and COX pathways was made in 1993 by Salvemini and coworkers [1]. They demonstrated that nitric oxide activated the COX enzymes, leading to overt production of PGs. This in turn exacerbated the inflammatory response. Such studies raised the possibility that COX

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Role of nitric oxide in chronic gut inflammation, the initiation and pathogenesis of these diseases are multi-factorial involving interactions among genetic, environmental and immune factors [1]. Regardless of exactly how these interactions ultimately promote chronic gut inflammation, it is becoming increasingly apparent that the immune system pla

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Roles of nitric oxide, superoxide, and peroxynitrite in myocardial ischemia-reperfusion injury and great clinical importance. The heart was also found to have an inherent ability to adapt to ischemie stress called ischemie preconditioning (PC) [1]. It is a well described adaptive response in which brief exposure of the heart to brief episode(s) of ischemia (PC ischemia) markedly enhances its abi

有特色

Regulation of the inducible nitric oxide synthase (iNOS) gene,r of human diseases and cancers [1–4]. L-arginine is converted to the NO and L-citrulline by the inducible isoform of nitric oxide synthase and produces high levels of NO (Fig. 1). Once produced, NO has a short half-life (t. = s) and undergoes spontaneous oxidation to the inactive metabolites nitrit

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疏忽

Direct and indirect modulation of the inducible nitric oxide synthase by nitric oxide: feedback mecanscriptional, post-transcriptional, translational, and post-translational levels. Cytokine activities interact with one another, regulate each other’s expression and activity, and baffle investigators with their often-overlapping spectra of action [1]. Cytokines are required to mediate many cellula

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Nitric oxide regulation of eicosanoid production,. Cyclooxygenase (COX), converts arachidonic acid to the prostaglandins (PG), prostacyclin (PGI.) and thromboxane A. (TXA.). Two major forms of NOS and COX have been identified to date. Under normal circumstances, the constitutive isoforms of these enzymes are found in virtually all organs. Their pr