Titlebook: New Horizons for Failing Heart Syndrome; Shigetake Sasayama Book 1996 Springer Japan 1996 Failing Heart Syndrome.circulation.heart.heart f

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Cytoskeletal Changes in the Calcium-Overloaded Heartrotubule disruption. Potent β3-adrenergic stimulation also disrupts microtubules of the myocardium in vitro and in vivo. Although microtubules do not directly contribute to the contractile function of the heart, microtubular disruption may cause cellular dysfunction and could be a precipitating fact

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Two Major Pumps of the Heart: Difference and Interdependence of the Right and Left Ventriclesolic and diastolic functions. Thus the structural and hemodynamic differences of these two pumping chambers determine the perfusion pressure-flow function integrity of the regional myocardium and the distribution of myocardial necrosis. Understanding the interdependence of these chambers will allow

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Dahl Salt-Sensitive Rats: Model to Study Transitions from Compensatory Hypertrophy to Left Ventriculght with a slope that was less steep in the failing DS rats than in age-matched DR rats. Therefore the DS rat is a useful model for demonstrating rapidly developing congestive heart failure, in which the transition from compensatory hypertrophy to decompensatory dilatation of the left ventricle is c

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Mechanoenergetics of Acute Failing Hearts Characterized by Oxygen Costs of Mechanical Energy and ConPVA and Emax, different from the above stunned and acidotic hearts. Emax in the second type of pathological heart was depressed comparably to the stunned and acidotic hearts, but its depression is considered due to a decreased total amount of Ca. handled during excitation-contraction (E-C) coupling.

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Left Ventricular Contractile and Energetic Dysfunction After Recovery from Acidosisthe postacidotic failing heart requires higher Vo. for a unit increase in Emax than the normal heart, which energetically resembles postischemic stunned myocardium. When dimethylamiloride, a selective Na.-H. exchange inhibitor, was administered just before the correction of acidosis, the development

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Diagnostic Value of the Transient Response of Oxygen Consumption to Exercise in Cardiac Patients: Rahe time constant of the slow monotonic rise was longer in patients than in control subjects (48 ± 37 versus 31 ± 8 s; . 0.005). In patients with mitral stenosis the time constant was longer (62 ± 23 s; . 0.005) with a small amplitude (195 ± 76 ml/min; . 0.05). Successful percutaneous transvenous mit

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Cytokines and Heart Failure: Immunomodulating Agents for the Management of Heart Failuree were markedly improved by treatment with vesnarinone. Vesnarinone inhibited the increase in natural killer cell activity and production of TNF-α in this animal model. We also studied the effects of vesnarinone on cytokine production by lipopolysaccharide-stimulated whole blood from patients with h