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Titlebook: New Cytokines as Potential Drugs; Satwant K. Narula,Robert Coffman Conference proceedings 2000 Springer Basel AG 2000 Chemokine.clinical r

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書(shū)目名稱(chēng)New Cytokines as Potential Drugs
編輯Satwant K. Narula,Robert Coffman
視頻videohttp://file.papertrans.cn/665/664972/664972.mp4
叢書(shū)名稱(chēng)Progress in Inflammation Research
圖書(shū)封面Titlebook: New Cytokines as Potential Drugs;  Satwant K. Narula,Robert Coffman Conference proceedings 2000 Springer Basel AG 2000 Chemokine.clinical r
描述Over the past ten years, a number of cytokines and growth factors have proven to be as effective therapeutics. While these products have certainly established recombinant biologics as a major pharmaceutical growth sector, the continued interest in this class of drugs arises from the fact that today we have a far better understanding of the human immune response, both at a cellular and molecular level. This has resulted in a more methodical characterisation of these factors which has given clinical researchers an opportunity to plan Phase 1 clinical trials that can provide substantial information on the activity of the cytokine in humans. Currently, a great deal of effort is also being channelled into identifying cytokines from the various DNA databases. Our major objective for this book is to profile cytokines that have been recently identified. The therapeutic potential of these cytokines based on their known properties will be discussed by the authors. The main aim of this book is to provide...
出版日期Conference proceedings 2000
關(guān)鍵詞Chemokine; clinical research; clinical trial; cytokine; immune response; research
版次1
doihttps://doi.org/10.1007/978-3-0348-8456-3
isbn_softcover978-3-0348-9575-0
isbn_ebook978-3-0348-8456-3Series ISSN 1422-7746 Series E-ISSN 2296-4525
issn_series 1422-7746
copyrightSpringer Basel AG 2000
The information of publication is updating

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Progress in Inflammation Researchhttp://image.papertrans.cn/n/image/664972.jpg
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Interleukin 17,. . and . studies suggest a proinflammatory function for this Interleukin but no data firmly establishes the status of IL-17 as the molecule to target in order to control the inflammatory response. The scope of this chapter is therefore a panoramic view of the pathologies where IL-17 appears to be involved.
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Interleukin 16,m mitogen-stimulated human peripheral blood mononuclear cells [1, 2]. IL-16 was originally designated as lymphocyte chemoattractant factor (LCF), ascribing to its first identified function of the induction of T lymphocyte migration. Since that initial observation the structure, mechanism of synthesi
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