Titlebook: Neurotransmitter Receptors; Mechanisms of Action Shozo Kito,Tomio Segawa,Richard W. Olsen Book 1984 The Editor(s) (if applicable) and The A

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Neuromodulatory Roles of Adenosine Receptors Coupling to the Calcium Channel and Adenylate Cyclasel stimulation. The released ATP is degraded to adenosine extracellularly. Adenosine derivatives also might be released from excited postsynaptic neurons. Therefore, during and after stimulation, adenosine derivatives are accumulated in the synaptic cleft. Physiological functions of ATP and adenosine

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Biochemical Properties of the GABA/Barbiturate/Benzodiazepine Receptor-Chloride Ion Channel Complexannels are regulated via GABA binding to its receptor. The cellular response to GABA has been shown to be enhanced by benzodiazepine and barbiturate drugs. This potentiation of GABA-mediated inhibition may explain much of the nervous system depressant action of these clinically important agents.

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Purification of γ-Aminobutyric Acid (GABA) and Benzodiazepine Receptors from Rat Brain Using Benzodiebrate nervous systems (5,6). GABA acts through a physiologically relevant receptor protein, the GABA receptor, which is labeled specifically by [.H]GABA (7) and various GABA agonists such as [.H]muscimol (8,9). On the other hand, it has been demonstrated that specific and pharmacologically relevant

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Structure and Properties of the Brain GABA/Benzodiazepine Receptor Complexrst is the GABA-A receptor, at which benzodiazepines potentiate the electrophysiological activity of GABA, bicuculline is a strong antagonist and muscimol, isoguvacine and certain other ligands are strong agonists. The second is the GABA-B receptor (1) which has none of the properties just mentioned

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