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Titlebook: Neurosciences at the Postgenomic Era; Jacques Mallet,Yves Christen Conference proceedings 2003 Springer-Verlag Berlin Heidelberg 2003 brai

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樓主: coerce
21#
發(fā)表于 2025-3-25 04:08:13 | 只看該作者
22#
發(fā)表于 2025-3-25 10:24:38 | 只看該作者
S. G. N. Grant,Thomas J. O’Dell President of the IEEE Robotics and Automation Society, and a Fellow of both IEEE and ASME. .Oussama Khatib is Professor at the prestigious Stanford University in the USA, President of the International Foundation of Robotics Research, and a recipient of the Japan Robot Association Award in Research
23#
發(fā)表于 2025-3-25 12:35:48 | 只看該作者
K. Mirnics,F. A. Middleton,J. N. Pierri,D. A. Lewis,Pat Levitt President of the IEEE Robotics and Automation Society, and a Fellow of both IEEE and ASME. .Oussama Khatib is Professor at the prestigious Stanford University in the USA, President of the International Foundation of Robotics Research, and a recipient of the Japan Robot Association Award in Research
24#
發(fā)表于 2025-3-25 16:37:19 | 只看該作者
25#
發(fā)表于 2025-3-25 20:16:40 | 只看該作者
26#
發(fā)表于 2025-3-26 01:08:51 | 只看該作者
27#
發(fā)表于 2025-3-26 05:13:50 | 只看該作者
0945-6082 e fields of speech and language disorders, X-linked mental retardation, gene therapy in the CNS, memory and learning disorders and other fields. .978-3-642-62430-8978-3-642-55543-5Series ISSN 0945-6082 Series E-ISSN 2196-3096
28#
發(fā)表于 2025-3-26 10:33:22 | 只看該作者
Analysis of Brain Disorders Using DNA Microarrays,hrenia, depression, autism or other mental disorders. In turn, this approach will lead to the improved application of pharmacogenomics, where compounds are screened for their ability to modulate the transcripts that are changed in a diagnosis-specific manner
29#
發(fā)表于 2025-3-26 15:29:15 | 只看該作者
30#
發(fā)表于 2025-3-26 20:26:59 | 只看該作者
Genetics and Physiopathology of X-linked Mental Retardation,t have a role in chromatin remodelling are affected in three important syndromic forms, while defects in signal transduction pathways implicated in neuronal maturation were found in “nonspecific” forms. These findings provide important insights into the molecular and cellular defects that underlie mental retardation.
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