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Titlebook: Neuroinflammation, Gut-Brain Axis and Immunity in Neuropsychiatric Disorders; Yong-Ku Kim Book 2023 The Editor(s) (if applicable) and The

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發(fā)表于 2025-3-21 16:18:48 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Neuroinflammation, Gut-Brain Axis and Immunity in Neuropsychiatric Disorders
編輯Yong-Ku Kim
視頻videohttp://file.papertrans.cn/665/664112/664112.mp4
概述Examines interactions between cytokines, glia, and neurons in the pathophysiology of neuropsychiatric disorders.Considers the role of inflammation in numerous disorders.Examines potential new targets
叢書(shū)名稱Advances in Experimental Medicine and Biology
圖書(shū)封面Titlebook: Neuroinflammation, Gut-Brain Axis and Immunity in Neuropsychiatric Disorders; Yong-Ku Kim Book 2023 The Editor(s) (if applicable) and The
描述.This book reviews the relationship between cytokines, glia, and neurons in the pathophysiology of neuropsychiatric disorders and examines the mechanisms of action of the drugs used for the treatment of these disorders. Increasing evidence has suggested that glia perform important roles in various brain functions, but much remains to be learned about.?.these crucial cells and their interplay with neurons. In addition, a better understanding of the interaction between inflammatory mediators, such as cytokines,.?.and the activated immune response will be of critical importance for the development of new therapeutic strategies. These key areas are the focus of this book, which documents the latest research findings in the field. Evidence is provided for the role of inflammation-induced toxic metabolites from the tryptophan pathway in a wide range of neuropsychiatric disorders, including depression, schizophrenia, and Alzheimer‘s disease. In presenting state of the art knowledge on the interactions between cytokines, glia, and neurons, the book will help to pave the way for the development of novel targets for the prevention and treatment of neuropsychiatric disorders..
出版日期Book 2023
關(guān)鍵詞glia; neuron; neuroinflammation; cytokine; depression; schizophrenia; bipolar disorder; dementia; anxiety; br
版次1
doihttps://doi.org/10.1007/978-981-19-7376-5
isbn_softcover978-981-19-7378-9
isbn_ebook978-981-19-7376-5Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Singapor
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C-Reactive Protein (CRP): A Potent Inflammation Biomarker in Psychiatric Disordershypothesis of the need for a different clinical and prognostic characterization based on this concomitant neuroinflammatory predisposition. However, even though further research studies are needed to confirm this preliminary evidence, CRP may represent a potential clinical routine biomarker which co
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Glial-Neuronal Interaction in Synapses: A Possible Mechanism of the Pathophysiology of Bipolar Disorion and structural changes of glial cells play a central role in the BD pathophysiology. This book chapter highlighted the latest findings that support the theory of synaptic dysfunction in BD, providing an overview of the alterations in neurotransmitters release, astrocytic uptake, and receptor sig
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PTSD, Immune System, and Inflammationflammation and disrupted cognition support the search for novel strategies for treatment of PTSD. Besides potential anti-inflammatory treatment, dietary interventions or the use of beneficial bacteria, such as probiotics, can potentially improve the composition and the function of the bacterial comm
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Sleep Immune Cross Talk and Insomniah insomnia disorder. These studies suggest that insomnia disorder is related to deregulation of the immune system, with an increase in the level of pro-inflammatory cytokines and change in rate of secretion and a decrease in the level of lymphocyte. Insomnia treatments, particularly cognitive behavi
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