Titlebook: Neuroendocrinology of Gastrointestinal Ulceration; Sandor Szabo,Yvette Taché,Gary B. Glavin Book 1995 Springer Science+Business Media New

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書目名稱Neuroendocrinology of Gastrointestinal Ulceration影響因子(影響力)




書目名稱Neuroendocrinology of Gastrointestinal Ulceration影響因子(影響力)學(xué)科排名




書目名稱Neuroendocrinology of Gastrointestinal Ulceration網(wǎng)絡(luò)公開度




書目名稱Neuroendocrinology of Gastrointestinal Ulceration網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Neuroendocrinology of Gastrointestinal Ulceration被引頻次




書目名稱Neuroendocrinology of Gastrointestinal Ulceration被引頻次學(xué)科排名




書目名稱Neuroendocrinology of Gastrointestinal Ulceration年度引用




書目名稱Neuroendocrinology of Gastrointestinal Ulceration年度引用學(xué)科排名




書目名稱Neuroendocrinology of Gastrointestinal Ulceration讀者反饋




書目名稱Neuroendocrinology of Gastrointestinal Ulceration讀者反饋學(xué)科排名

龍蝦

Overview: 978-1-4613-5759-9978-1-4615-1867-9

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Corticotropin Releasing Factor and Gastric ErosionsSeveral neuropeptides have been shown to either promote or inhibit the development of gastric erosions in rats; recent reviews have been presented by Taché. and Hernandez..

敲詐

Role of the Vagal Nerve in the Development of Gastric Mucosal Injury and its Prevention by Atropine,Peptic ulceration including acute erosions is a multifactorial disease involving well-known factors such as trauma, stress, sepsis, hemorrhagic shock, burns, pulmonary and liver diseases. and drugs such as reserpine., epinephrine., nonsteroidal antiinflammatory drugs., steroids and other chemicals..

兩種語言

The Effect of bFGF and PDGF on Acute Gastric Mucosal Lesions, Chronic Gastritis and Chronic Duodenalstric acid or inhibiting acid secretion. In these cases the erosions and ulcers were essentially left alone to heal themselves in the presence of diminished aggressive factors, but without any attempt of stimulating the healing process.

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Hans Selye Symposia on Neuroendocrinology and Stresshttp://image.papertrans.cn/n/image/664030.jpg

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Role of Sensory Neurons in the Control of Gastric Mucosal Blood Flow and Protection and of both endogenous and exogenous origin. Most tissues would rapidly disintegrate if exposed to the concentrations of hydrochloric acid that bathe but do not harm the gastric surface epithelium. This is because a multitude of mechanisms constituting the gastric mucosal barrier prevents hydrogen

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