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Titlebook: Neurodegenerative Diseases; Clinical Aspects, Mo Daniela Galimberti,Elio Scarpini Book 20141st edition Springer-Verlag London 2014 Alzheime

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發(fā)表于 2025-3-21 19:22:06 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Neurodegenerative Diseases
副標(biāo)題Clinical Aspects, Mo
編輯Daniela Galimberti,Elio Scarpini
視頻videohttp://file.papertrans.cn/664/663978/663978.mp4
概述Covers clinical and genetic aspects, as well as biomarkers for early diagnosis and treatment.Translational (from bench to bedside) approach.Includes chapters on neurodegeneration in classically non-de
圖書封面Titlebook: Neurodegenerative Diseases; Clinical Aspects, Mo Daniela Galimberti,Elio Scarpini Book 20141st edition Springer-Verlag London 2014 Alzheime
描述.This book gives an overview of the current knowledge on the most common neurodegenerative diseases, including Alzheimer’s disease, frontotemporal lobar degeneration, amyotrophic lateral sclerosis, and additional neurodegenerative diseases. Different aspects of each disease are reviewed, including clinical issues, treatments, basic discoveries (genetics and molecular biology), and translation of basic research into biomarkers for early diagnosis. In addition, emerging data indicate that neurodegeneration seems to also be present in classically non-degenerative disorders. Therefore, a chapter about overlapping mechanisms between dementias and psychiatric disorders is included, as well as a description of the role of neurodegeneration in multiple sclerosis..Neurodegenerative Diseases. is aimed at clinicians, particularly those working in academic hospitals. This multidisciplinary book will also be of interest to basic researchers in medical fields.? .
出版日期Book 20141st edition
關(guān)鍵詞Alzheimer‘s; Amyotrophic Lateral Sclerosis; Frontotemporal Lobar Degeneration; dementia; neurodegenerati
版次1
doihttps://doi.org/10.1007/978-1-4471-6380-0
isbn_softcover978-1-4471-7242-0
isbn_ebook978-1-4471-6380-0
copyrightSpringer-Verlag London 2014
The information of publication is updating

書目名稱Neurodegenerative Diseases影響因子(影響力)




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沙發(fā)
發(fā)表于 2025-3-21 20:52:49 | 只看該作者
板凳
發(fā)表于 2025-3-22 01:48:45 | 只看該作者
Book 20141st editionar degeneration, amyotrophic lateral sclerosis, and additional neurodegenerative diseases. Different aspects of each disease are reviewed, including clinical issues, treatments, basic discoveries (genetics and molecular biology), and translation of basic research into biomarkers for early diagnosis.
地板
發(fā)表于 2025-3-22 06:01:14 | 只看該作者
5#
發(fā)表于 2025-3-22 11:03:58 | 只看該作者
,Alzheimer’s Disease and Frontotemporal Lobar Degeneration: Mouse Models,have seen the identification of new genes involved in the disease. This led to the generation of a large number of new transgenic mouse models for FTLD. This chapter provides an overview of APP and tau-based mouse models of AD and FTLD and discusses in detail the more recent FTLD models expressing novel disease genes.
6#
發(fā)表于 2025-3-22 13:17:23 | 只看該作者
Amyotrophic Lateral Sclerosis: Genotypes and Phenotypes,notypes. This information is essential for biologists and clinicians alike, providing at the same time an unparalleled insight into the pathogenesis of the disease and invaluable tools for genetic counseling, diagnosis, and development of preventive strategies and treatments for ALS.
7#
發(fā)表于 2025-3-22 20:55:24 | 只看該作者
Neurodegeneration and Multiple Sclerosis,d Parkinson’s disease, as well as multiple sclerosis (MS), diabetes, acute brain injury among many other conditions. This chapter discusses the clinical and pathophysiological features of neurodegeneration in MS.
8#
發(fā)表于 2025-3-22 22:07:29 | 只看該作者
9#
發(fā)表于 2025-3-23 01:24:22 | 只看該作者
,Alzheimer’s Disease: Clinical Aspects and Treatments,biological biomarkers, in particular radiological and cerebrospinal fluid Amyloid, tau and phospho-tau levels. Here, the main clinical aspects and diagnostic tools for AD are revised; atypical AD presentations and possible diagnostic pitfalls are also discussed.
10#
發(fā)表于 2025-3-23 09:16:45 | 只看該作者
,Autosomal Dominant Alzheimer’s Disease: Underlying Causes,rk (DIAN). We provide an overview for the molecular genetic findings available for causal AD genes, discuss how this knowledge can be applied in clinical practice, and highlight the strategies to detect novel AD genes (e.g., . and .).
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