Titlebook: Neurodegenerative Diseases; Pathology, Mechanism Philip Beart,Michael Robinson,Nicholas J. Maragaki Book 2017 Springer International Publis

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書目名稱Neurodegenerative Diseases影響因子(影響力)




書目名稱Neurodegenerative Diseases影響因子(影響力)學(xué)科排名




書目名稱Neurodegenerative Diseases網(wǎng)絡(luò)公開度




書目名稱Neurodegenerative Diseases網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Neurodegenerative Diseases被引頻次




書目名稱Neurodegenerative Diseases被引頻次學(xué)科排名




書目名稱Neurodegenerative Diseases年度引用




書目名稱Neurodegenerative Diseases年度引用學(xué)科排名




書目名稱Neurodegenerative Diseases讀者反饋




書目名稱Neurodegenerative Diseases讀者反饋學(xué)科排名

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Neurodegenerative Diseases978-3-319-57193-5Series ISSN 2190-5215 Series E-ISSN 2190-5223

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Clinical Aspects of Alzheimer’s Diseaseimer’s disease and provides an overview of the clinical syndromes associated with Alzheimer pathology and their associated neuroimaging findings. This chapter also reviews the neuropathology and genetics of Alzheimer’s disease and concludes by discussing current work undertaken to identify suitable in vivo biomarkers for the disease.

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Philip Beart,Michael Robinson,Nicholas J. MaragakiDiscusses molecular and cellular aspects of neurodegeneration in a contemporary context.Includes both basic research and clinical perspectives.Evaluates modern research methods and contributions

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Clinical Aspects of Alzheimer’s Diseaseimer’s disease and provides an overview of the clinical syndromes associated with Alzheimer pathology and their associated neuroimaging findings. This chapter also reviews the neuropathology and genetics of Alzheimer’s disease and concludes by discussing current work undertaken to identify suitable

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Parkinson’s Disease: Basic Pathomechanisms and a Clinical Overviewisease. The majority of existing treatments target motor symptom control. Over the last two decades the impact of the genetic contribution to PD has been appreciated. Significant discoveries have been made, which have advanced our understanding of the pathophysiological and molecular basis of PD. In

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Huntington’s Disease: Pathogenic Mechanisms and Therapeutic Targetsptoms, cognitive deficits culminating in dementia, and the movement disorder epitomised by motor signs such as chorea. HD is caused by a CAG repeat expansion encoding an extended tract of the amino acid glutamine in the huntingtin protein. This polyglutamine expansion appears to induce a ‘change of