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Titlebook: Neurochemical Mechanisms in Disease; John P. Blass Book 2011 Springer Science+Business Media, LLC 2011

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發(fā)表于 2025-3-21 19:52:34 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Neurochemical Mechanisms in Disease
編輯John P. Blass
視頻videohttp://file.papertrans.cn/664/663936/663936.mp4
概述Addresses the neurochemistry of disease through a combination of diseases and disease sources.Includes chapters on both human diseases and animal “model” diseases.Written by experts in the field.Inclu
叢書名稱Advances in Neurobiology
圖書封面Titlebook: Neurochemical Mechanisms in Disease;  John P. Blass Book 2011 Springer Science+Business Media, LLC 2011
描述This newest volume of Advances in Neurobiology deals with the Neurochemistry of disease, with chapters covering both human diseases and animal “model” diseases.
出版日期Book 2011
版次1
doihttps://doi.org/10.1007/978-1-4419-7104-3
isbn_softcover978-1-4939-4085-1
isbn_ebook978-1-4419-7104-3Series ISSN 2190-5215 Series E-ISSN 2190-5223
issn_series 2190-5215
copyrightSpringer Science+Business Media, LLC 2011
The information of publication is updating

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Animal Models of Neurodegenerative Diseases,sed by these studies is the growing necessity to standardize the experimental procedures used to obtain an animal model, housing and breeding conditions, assessments of phenotypes investigated and, ultimately the interpretation of results obtained and their relevance to the pathology.
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Brain Edema in Neurological Diseases,adicals, and products of the arachidonic acid metabolism. We review present knowledge of the contribution to brain edema of molecules such as aquaporins, vasopressin, vascular endothelial growth factor, angiopoietins, and bradykinin. We further examine brain imaging modalities that have revolutioniz
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Rho-Linked Mental Retardation Genes, as to how defective cellular signaling can result in abnormal neuronal connectivity, which can give rise to impaired information processing underlying cognitive function. Aberrations in defined Rho-mediated signaling pathways have been linked to defects in the formation and remodeling of dendritic
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Trinucleotide-Expansion Diseases,sed by (CAG). expansions, the mutation is in an exon of the gene and each protein is expressed with an expanded polyglutamine (Q.) domain. At least nine neurodegenerative diseases belong in this second group. The most common of these diseases is Huntington disease (HD). Others are dentatorubralpalli
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CNS Cytokines,ive diseases, such as Alzheimer’s disease, Parkinson’s disease, multiple sclerosis, and cerebral palsy. This chronic neuroinflammation is associated with a number of common factors; most notable among these is the increased concentration of proinflammatory cytokines. In addition to the ones listed a
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Neurochemistry of Endogenous Antinociception,fect of a ligand is determined by the activation/inhibition of the different types of receptors and the location of these receptors, however, only a part of the endogenous substances has been characterized extensively in this respect.
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