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Titlebook: Neurochemical Aspects of Neurotraumatic and Neurodegenerative Diseases; Akhlaq A. Farooqui Book 2010 Springer Science+Business Media, LLC

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發(fā)表于 2025-3-21 18:00:20 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Neurochemical Aspects of Neurotraumatic and Neurodegenerative Diseases
編輯Akhlaq A. Farooqui
視頻videohttp://file.papertrans.cn/664/663934/663934.mp4
概述Presents readers with cutting edge and comprehensive information on molecular aspects of acute neural trauma and neurodegenerative diseases.This book can be used as supplement text for a range of neur
圖書(shū)封面Titlebook: Neurochemical Aspects of Neurotraumatic and Neurodegenerative Diseases;  Akhlaq A. Farooqui Book 2010 Springer Science+Business Media, LLC
描述Collectively, neurodegenerative diseases are characterized by chronic and progressive loss of neurons in discrete areas of the brain, producing debilitating symptoms such as dementia, loss of memory, loss of sensory or motor capability, decreased overall quality of life eventually leading to premature death. Two types of cell death are known to occur during neurodegeneration: (a) apoptosis and (b) necrosis. The necrosis is characterized by the passive cell swelling, intense mitochondrial damage with rapid loss of ATP, alterations in neural membrane permeability, high calcium influx, and disruption of ion homeostasis.This type of cell death leads to membrane lysis and release of intracellular components that induce inflammatory reactions.Necrotic cell death normally occurs at the core of injury site.In contrast, apoptosis is an active process in which caspases (a group of endoproteases with specificity for aspartate residues in protein) are stimulated.Apoptotic cell death is accompanied by cell shrinkage, dynamic membrane blebbing, chromatin condensation, DNA laddering, loss of phospholipids asymmetry, low ATP levels, and mild calcium overload.This type of cell death normally occurs
出版日期Book 2010
關(guān)鍵詞Alzheimer; Neurodegeneration; Parkinson; acute neural trauma; excitotoxicity; neurons
版次1
doihttps://doi.org/10.1007/978-1-4419-6652-0
isbn_softcover978-1-4899-8256-8
isbn_ebook978-1-4419-6652-0
copyrightSpringer Science+Business Media, LLC 2010
The information of publication is updating

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發(fā)表于 2025-3-21 21:35:27 | 只看該作者
Neurodegeneration in Neural Trauma, Neurodegenerative Diseases, and Neuropsychiatric Disorders,clin-dependent kinases) and upregulated anti-apoptotic mechanisms such as neurotrophic factor signaling, antioxidant enzymes, protein chaperones, anti-apoptotic proteins, and ionostatic systems (Nguyen et al., 2002).
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Potential Neuroprotective Strategies for Ischemic Injury,on, memory loss, language deficits, emotional difficulties, pain, and depression. About 4.7 million stroke survivors currently live in the USA. The number of stroke patients is expected to increase worldwide as the population continues to age.
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This book can be used as supplement text for a range of neurCollectively, neurodegenerative diseases are characterized by chronic and progressive loss of neurons in discrete areas of the brain, producing debilitating symptoms such as dementia, loss of memory, loss of sensory or motor capability, dec
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Neurochemical Aspects of Spinal Cord Injury,raumatic insult (Klussmann and Martin-Villalba, 2005). Unlike the primary event, which is instantaneous and beyond therapeutic management, the secondary event develops over the hours and days after SCI, causing behavioral and functional impairments.
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Potential Neuroprotective Strategies for Traumatic Brain Injury,he, confusion, lightheadedness, dizziness, blurred vision, fatigue, and trouble with memory (Bahraini et al., 2009). Moderate TBI produces a headache that gets worse with time, seizures, inability to awaken from sleep, dilation of one or both pupils of the eyes, slurred speech, loss of coordination, increased confusion.
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Neurochemical Aspects of Traumatic Brain Injury,on after TBI. Most common causes of TBI are car accidents, bicycle accidents (more than 50%), falls and sport injuries (20–25%), and violence and domestic abuse (including shaken baby syndrome) (20–25%). TBI produces physical, cognitive, emotional, and behavioral effects in the traumatized subject.
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