Titlebook: Neural Development and Schizophrenia; Theory and Research Sarnoff A. Mednick,J. Meggin Hollister Book 1995 Springer Science+Business Media

拼圖游戲

書(shū)目名稱Neural Development and Schizophrenia影響因子(影響力)




書(shū)目名稱Neural Development and Schizophrenia影響因子(影響力)學(xué)科排名




書(shū)目名稱Neural Development and Schizophrenia網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱Neural Development and Schizophrenia網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱Neural Development and Schizophrenia被引頻次




書(shū)目名稱Neural Development and Schizophrenia被引頻次學(xué)科排名




書(shū)目名稱Neural Development and Schizophrenia年度引用




書(shū)目名稱Neural Development and Schizophrenia年度引用學(xué)科排名




書(shū)目名稱Neural Development and Schizophrenia讀者反饋




書(shū)目名稱Neural Development and Schizophrenia讀者反饋學(xué)科排名

puzzle

燈絲

Distribution

audiologist

phrenia. The first was sponsored by the Schiwphrenia Research Branch with the scientific cooperation of Dr. David Shore. We met in Washington; the output of the meeting was published in a book entitled, Fetal Neural Development and Adult Schizophrenia. Cambridge University Press. 1991. The next meet

蹣跚

狼群

Fetal Development and Schizophrenia: Historical Observations from Teratology produces an error in development of her fetus which eventually results in schizophrenia in the child, now grown into a young adult. This hypothesis implicitly assumes several points. (1) Because the symptoms of schizophrenia are behavioral, we assume that the organ of the fetus which is finally aff

祖先

Dorsal-Kyphosis

Childhood Risk Factors for Adult Schizophrenia in a General Population Birth Cohort at Age 43 Yearsarker et al., 1989; Barker et al., 1990; Godfrey et al., 1993; Fine et al., 1985; Barker & Osmond, 1986; Colley et al., 1973; Martyn et al., 1988). Mechanisms which may explain these child-adult continuities include the programming thought to occur following impaired fetal nutrition and growth (Posk

艦旗

Subtypes of Schizophrenia: Diagnostic and Conceptual Issuesrenia” in 1911, controversy has raged over the question of whether the diverse clinical manifestations of schizophrenia are all derived from a single underlying etiopathological process or, alternatively, clinical subtypes of schizophrenia can be satisfactorily differentiated and linked to two or mo