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Titlebook: Nephrotoxic Mechanisms of Drugs and Environmental Toxins; George A. Porter Book 1982 Springer Science+Business Media New York 1982 Ketone.

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書目名稱Nephrotoxic Mechanisms of Drugs and Environmental Toxins
編輯George A. Porter
視頻videohttp://file.papertrans.cn/663/662676/662676.mp4
圖書封面Titlebook: Nephrotoxic Mechanisms of Drugs and Environmental Toxins;  George A. Porter Book 1982 Springer Science+Business Media New York 1982 Ketone.
描述The majority of the offending toxicants to be reviewed in this volume were devel- oped to help mankind, and it is only with prolonged or widespread application that their adverse effects have been recognized. Conversely, in the case of pre scrip- tion drugs, there has been an attempt to identify the adverse effects in advance and incorporate these risks into the decision of approval for human consumption. Unfortunately, for those drugs in which recognized injury occurs only after prolonged use, such appraisals are made in retrospect. Despite this, most renal injury induced by drugs or toxicants can be either prevented by excluding drugs with unacceptable side effects or interrupted by eliminating the offending agent once damage is manifested. The fact that prevention, reversibility, or arrest of renal injury is possible provided a major impetus for this publication. Since no international registry for nephrotoxic injury exists, estimates of incidence must rely on less than ideal sources. Recently I, together with Dr. William Bennett, summarized a survey of the frequency of various categories of nephrologic disease (Porter and Bennett, 1981). Based on this survey, we projected that
出版日期Book 1982
關(guān)鍵詞Ketone; Pet; dialysis; environment; etiology; iron; natural product; toxicity
版次1
doihttps://doi.org/10.1007/978-1-4684-4214-4
isbn_softcover978-1-4684-4216-8
isbn_ebook978-1-4684-4214-4
copyrightSpringer Science+Business Media New York 1982
The information of publication is updating

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Nephrotoxicity of Cephalosporin Antibioticse enough with some to restrict their use significantly, but ranges from relatively mild to absent with others (Tune and Fravert, 1980b). The reasons for these individual differences have not been fully elucidated.
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espread application that their adverse effects have been recognized. Conversely, in the case of pre scrip- tion drugs, there has been an attempt to identify the adverse effects in advance and incorporate these risks into the decision of approval for human consumption. Unfortunately, for those drugs
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Studies on Segmental Transport in Models of Acute Renal Failureevel of the individual nephron segment. The effect of ischemia on segmental nephron function was assessed by the method of isolated tubule microperfusion. The response of nephron segments to ischemic injury provides a rational explanation for the genesis of the syndrome and the laboratory values encountered.
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Amphotericin B Toxicity for Epithelial Cellse aminoglycosides appear to be intracellular organelles, i.e., lysosomes, in the proximal tubular cells. In most instances, the precise sequence of events leading to injury and functional impairment remains unknown.
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