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Titlebook: Natural Resistance Mechanisms of Plants to Viruses; Gad Loebenstein,John Peter Carr Book 2006 Springer Science+Business Media B.V. 2006 Br

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發(fā)表于 2025-3-21 17:10:48 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Natural Resistance Mechanisms of Plants to Viruses
編輯Gad Loebenstein,John Peter Carr
視頻videohttp://file.papertrans.cn/662/661913/661913.mp4
概述Links classical biological information with recent molecular studies
圖書(shū)封面Titlebook: Natural Resistance Mechanisms of Plants to Viruses;  Gad Loebenstein,John Peter Carr Book 2006 Springer Science+Business Media B.V. 2006 Br
描述Over the course of evolution most plants have acquired the ability to defend themselves against most groups of pathogens, including the viruses. Many antiviral resistance phenomena have been known and studied for decades but, until recently, understanding of their underlying mechanisms has lagged behind. These phenomena include resistance to infection, resistance to virus translocation through the plant, recovery from infection and genetically defined resistance, together with the associated phenomena of the local lesion response, and induced, or acquired, resistance. The identification and cloning of plant resistance genes, characterization of downstream signaling components, and especially the explosion of data regarding gene-silencing mechanisms, has led to rapid progress in the investigation of natural resistance phenomena. Meanwhile, in plant virology there has been remarkable progress in the arenas of replication, movement proteins and plasmodesmatal gating, and in the discovery of gene silencing suppressors. Therefore, it seemed timely and appropriate to link older but still important data on the well known, ‘classical’ resistance phenomena with the new information that has
出版日期Book 2006
關(guān)鍵詞Brassicaceae; Gene silencing; Moore; Resistance mechanisms; Resistant varieties; metabolism
版次1
doihttps://doi.org/10.1007/1-4020-3780-5
isbn_softcover978-90-481-6963-4
isbn_ebook978-1-4020-3780-1
copyrightSpringer Science+Business Media B.V. 2006
The information of publication is updating

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Resistance to Infectionterm plant culture at elevated temperature (“thermotherapy”), chemotherapy or plant micropropagation. Preventing virus-plant contact may involve clean stock programs and/or at least minimal applications of pesticides to control virus vectors (.). Biologically based interference is considered in Chapters 1, 6 and 13 and here.
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Book 2006antiviral resistance phenomena have been known and studied for decades but, until recently, understanding of their underlying mechanisms has lagged behind. These phenomena include resistance to infection, resistance to virus translocation through the plant, recovery from infection and genetically de
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Induced Resistance Mechanismson physical barriers such as the cell wall or cuticle, or resistance engendered by pre-existing antimicrobial compounds (.). However, certain resistance mechanisms, most particularly those that are inducible, are complex in nature and have proved to be more difficult to understand, particularly with respect to resistance to viruses.
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Resistance to , in the , esistance genes (.), ecology in wild plants (.) and genetic diversity (.) it is the best characterised potyvirus. As the potyvirus best adapted to the model, fully sequenced dicot plant ., it represents an excellent model with which to gain fundamental insights into plant — virus interactions.
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Viral Determinants of Resistance Versus Susceptibilityto replicate in an individual cell, but its cell-to-cell movement protein may be non-functional in that host, thereby limiting its movement into adjacent cells. In this instance, there would be no visible response of the plant to the inoculation of the virus.
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