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Titlebook: Model Systems in Aging; Thomas Nystr?m,Heinz D. Osiewacz Book 20041st edition Springer-Verlag Berlin Heidelberg 2004 aging.free radicals.g

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書目名稱Model Systems in Aging
編輯Thomas Nystr?m,Heinz D. Osiewacz
視頻videohttp://file.papertrans.cn/636/635800/635800.mp4
概述The unique feature of this book is its comparative approach to different genetic model systems.Full text available online for subscribers of the series
叢書名稱Topics in Current Genetics
圖書封面Titlebook: Model Systems in Aging;  Thomas Nystr?m,Heinz D. Osiewacz Book 20041st edition Springer-Verlag Berlin Heidelberg 2004 aging.free radicals.g
描述.Aging is the progressive decline in biological functions over time. This decline targets macromolecules, cells, tissues and, as a consequence, whole organisms. Despite considerable progress in the development of testable hypothesis concerning aging in an evolutionary context, a unifying theory of the molecular/physiological mechanistic causes of aging has not been reached. In fact, is it not clear to what extent aging is a programmed or stochastic process. ..This book takes the reader from unicellular bacterial deterioration via senescence in fungi and worms to aging in rodents and humans, allowing a comparative view on similarities and differences in different genetic model systems. The different model systems are scrutinized in the light of contemporary aging hypothesis, such as the free radical and genomic instability theories. ..
出版日期Book 20041st edition
關(guān)鍵詞aging; free radicals; genetic model systems; stress; apoptosis; bacteria; biological functions; development
版次1
doihttps://doi.org/10.1007/978-3-540-37005-5
isbn_softcover978-3-642-05698-7
isbn_ebook978-3-540-37005-5Series ISSN 1610-2096 Series E-ISSN 1610-6970
issn_series 1610-2096
copyrightSpringer-Verlag Berlin Heidelberg 2004
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Book 20041st editionorganisms. Despite considerable progress in the development of testable hypothesis concerning aging in an evolutionary context, a unifying theory of the molecular/physiological mechanistic causes of aging has not been reached. In fact, is it not clear to what extent aging is a programmed or stochast
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978-3-642-05698-7Springer-Verlag Berlin Heidelberg 2004
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Model Systems in Aging978-3-540-37005-5Series ISSN 1610-2096 Series E-ISSN 1610-6970
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